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CaM Kinase II mediates maladaptive post-infarct remodeling and pro-inflammatory chemoattractant signaling but not acute myocardial ischemia/reperfusion injury
CaMKII was suggested to mediate ischemic myocardial injury and adverse cardiac remodeling. Here, we investigated the roles of different CaMKII isoforms and splice variants in ischemia/reperfusion (I/R) injury by the use of new genetic CaMKII mouse models. Although CaMKIIδC was upregulated 1 day afte...
Autores principales: | Weinreuter, Martin, Kreusser, Michael M, Beckendorf, Jan, Schreiter, Friederike C, Leuschner, Florian, Lehmann, Lorenz H, Hofmann, Kai P, Rostosky, Julia S, Diemert, Nathalie, Xu, Chang, Volz, Hans Christian, Jungmann, Andreas, Nickel, Alexander, Sticht, Carsten, Gretz, Norbert, Maack, Christoph, Schneider, Michael D, Gröne, Hermann-Josef, Müller, Oliver J, Katus, Hugo A, Backs, Johannes |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BlackWell Publishing Ltd
2014
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4287929/ https://www.ncbi.nlm.nih.gov/pubmed/25193973 http://dx.doi.org/10.15252/emmm.201403848 |
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