Toll-like receptor 3 activation is required for normal skin barrier repair following UV damage

Ultraviolet (UV) damage to the skin leads to the release of noncoding RNA (ncRNA) from necrotic keratinocytes that activates toll-like receptor 3 (TLR3). This release of ncRNA triggers inflammation in the skin following UV damage. Recently, TLR3 activation was also shown to aid wound repair and incr...

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Detalles Bibliográficos
Autores principales: Borkowski, Andrew W., Kuo, I-Hsin, Bernard, Jamie J., Yoshida, Takeshi, Williams, Michael R., Hung, Nai-Jung, Yu, Benjamin D., Beck, Lisa A., Gallo, Richard L.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: 2014
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4289479/
https://www.ncbi.nlm.nih.gov/pubmed/25118157
http://dx.doi.org/10.1038/jid.2014.354

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4289479/
https://www.ncbi.nlm.nih.gov/pubmed/25118157
http://dx.doi.org/10.1038/jid.2014.354

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