Alarmins released during local antitumor treatments play an essential role in enhancing tumor growth inhibition at treated and non-treated sites via a derivative of CCL3

ECI301 (eMIP), a single amino-acid substituted CCL3 (MIP-1α), enhanced tumor growth inhibition and the abscopal effect (an effect distal to the target) following local antitumor therapy such as radiation, radiofrequency ablation (RFA), or hyperthermia treatment. The recent elucidation of the underly...

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Autores principales: Kanegasaki, Shiro, Tsuchiya, Tomoko
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Taylor & Francis 2014
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4292266/
https://www.ncbi.nlm.nih.gov/pubmed/25941584
http://dx.doi.org/10.4161/21624011.2014.958956
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author Kanegasaki, Shiro
Tsuchiya, Tomoko
author_facet Kanegasaki, Shiro
Tsuchiya, Tomoko
author_sort Kanegasaki, Shiro
collection PubMed
description ECI301 (eMIP), a single amino-acid substituted CCL3 (MIP-1α), enhanced tumor growth inhibition and the abscopal effect (an effect distal to the target) following local antitumor therapy such as radiation, radiofrequency ablation (RFA), or hyperthermia treatment. The recent elucidation of the underlying mechanism may lead to a better antitumor therapy.
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spelling pubmed-42922662015-12-15 Alarmins released during local antitumor treatments play an essential role in enhancing tumor growth inhibition at treated and non-treated sites via a derivative of CCL3 Kanegasaki, Shiro Tsuchiya, Tomoko Oncoimmunology Article ECI301 (eMIP), a single amino-acid substituted CCL3 (MIP-1α), enhanced tumor growth inhibition and the abscopal effect (an effect distal to the target) following local antitumor therapy such as radiation, radiofrequency ablation (RFA), or hyperthermia treatment. The recent elucidation of the underlying mechanism may lead to a better antitumor therapy. Taylor & Francis 2014-12-15 /pmc/articles/PMC4292266/ /pubmed/25941584 http://dx.doi.org/10.4161/21624011.2014.958956 Text en © 2014 The Author(s). Published with license by Taylor & Francis Group, LLC http://creativecommons.org/licenses/by-nc/3.0/ This is an Open Access article distributed under the terms of the Creative Commons Attribution-Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The moral rights of the named author(s) have been asserted.
spellingShingle Article
Kanegasaki, Shiro
Tsuchiya, Tomoko
Alarmins released during local antitumor treatments play an essential role in enhancing tumor growth inhibition at treated and non-treated sites via a derivative of CCL3
title Alarmins released during local antitumor treatments play an essential role in enhancing tumor growth inhibition at treated and non-treated sites via a derivative of CCL3
title_full Alarmins released during local antitumor treatments play an essential role in enhancing tumor growth inhibition at treated and non-treated sites via a derivative of CCL3
title_fullStr Alarmins released during local antitumor treatments play an essential role in enhancing tumor growth inhibition at treated and non-treated sites via a derivative of CCL3
title_full_unstemmed Alarmins released during local antitumor treatments play an essential role in enhancing tumor growth inhibition at treated and non-treated sites via a derivative of CCL3
title_short Alarmins released during local antitumor treatments play an essential role in enhancing tumor growth inhibition at treated and non-treated sites via a derivative of CCL3
title_sort alarmins released during local antitumor treatments play an essential role in enhancing tumor growth inhibition at treated and non-treated sites via a derivative of ccl3
topic Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4292266/
https://www.ncbi.nlm.nih.gov/pubmed/25941584
http://dx.doi.org/10.4161/21624011.2014.958956
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AT tsuchiyatomoko alarminsreleasedduringlocalantitumortreatmentsplayanessentialroleinenhancingtumorgrowthinhibitionattreatedandnontreatedsitesviaaderivativeofccl3

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