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Cardiac resynchronization therapy for heart failure induced by left bundle branch block after transcatheter closure of ventricular septal defect
A 54-year-old female patient with congenital heart disease had a persistent complete left bundle branch block three months after closure by an Amplatzer ventricular septal defect occluder. Nine months later, the patient suffered from chest distress, palpitation, and sweating at daily activities, and...
Autores principales: | , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Science Press
2014
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4294153/ https://www.ncbi.nlm.nih.gov/pubmed/25593586 http://dx.doi.org/10.11909/j.issn.1671-5411.2014.04.012 |
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author | Du, Rong-Zeng Qian, Jun Wu, Jun Liang, Yi Chen, Guang-Hua Sun, Tao Zhou, Ye Zhao, Yang Yan, Jin-Chuan |
author_facet | Du, Rong-Zeng Qian, Jun Wu, Jun Liang, Yi Chen, Guang-Hua Sun, Tao Zhou, Ye Zhao, Yang Yan, Jin-Chuan |
author_sort | Du, Rong-Zeng |
collection | PubMed |
description | A 54-year-old female patient with congenital heart disease had a persistent complete left bundle branch block three months after closure by an Amplatzer ventricular septal defect occluder. Nine months later, the patient suffered from chest distress, palpitation, and sweating at daily activities, and her 6-min walk distance decreased significantly (155 m). Her echocardiography showed increased left ventricular end-diastolic diameter with left ventricular ejection fraction of 37%. Her symptoms reduced significantly one week after received cardiac resynchronization therapy. She had no symptoms at daily activities, and her echo showed left ventricular ejection fraction of 46% and 53%. Moreover, left ventricular end-diastolic diameter decreased 6 and 10 months after cardiac resynchronization therapy, and 6-min walk distance remarkably increased. This case demonstrated that persistent complete left bundle branch block for nine months after transcatheter closure with ventricular septal defect Amplatzer occluder could lead to left ventricular enlargement and a significant decrease in left ventricular systolic function. Cardiac resynchronization therapy decreased left ventricular end-diastolic diameter and increased left ventricular ejection fraction, thereby improving the patient's heart functions. |
format | Online Article Text |
id | pubmed-4294153 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2014 |
publisher | Science Press |
record_format | MEDLINE/PubMed |
spelling | pubmed-42941532015-01-20 Cardiac resynchronization therapy for heart failure induced by left bundle branch block after transcatheter closure of ventricular septal defect Du, Rong-Zeng Qian, Jun Wu, Jun Liang, Yi Chen, Guang-Hua Sun, Tao Zhou, Ye Zhao, Yang Yan, Jin-Chuan J Geriatr Cardiol Case Report A 54-year-old female patient with congenital heart disease had a persistent complete left bundle branch block three months after closure by an Amplatzer ventricular septal defect occluder. Nine months later, the patient suffered from chest distress, palpitation, and sweating at daily activities, and her 6-min walk distance decreased significantly (155 m). Her echocardiography showed increased left ventricular end-diastolic diameter with left ventricular ejection fraction of 37%. Her symptoms reduced significantly one week after received cardiac resynchronization therapy. She had no symptoms at daily activities, and her echo showed left ventricular ejection fraction of 46% and 53%. Moreover, left ventricular end-diastolic diameter decreased 6 and 10 months after cardiac resynchronization therapy, and 6-min walk distance remarkably increased. This case demonstrated that persistent complete left bundle branch block for nine months after transcatheter closure with ventricular septal defect Amplatzer occluder could lead to left ventricular enlargement and a significant decrease in left ventricular systolic function. Cardiac resynchronization therapy decreased left ventricular end-diastolic diameter and increased left ventricular ejection fraction, thereby improving the patient's heart functions. Science Press 2014-12 /pmc/articles/PMC4294153/ /pubmed/25593586 http://dx.doi.org/10.11909/j.issn.1671-5411.2014.04.012 Text en Institute of Geriatric Cardiology http://creativecommons.org/licenses/by-nc-sa/3.0/ This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 3.0 Unported License, which allows readers to alter, transform, or build upon the article and then distribute the resulting work under the same or similar license to this one. The work must be attributed back to the original author and commercial use is not permitted without specific permission. |
spellingShingle | Case Report Du, Rong-Zeng Qian, Jun Wu, Jun Liang, Yi Chen, Guang-Hua Sun, Tao Zhou, Ye Zhao, Yang Yan, Jin-Chuan Cardiac resynchronization therapy for heart failure induced by left bundle branch block after transcatheter closure of ventricular septal defect |
title | Cardiac resynchronization therapy for heart failure induced by left bundle branch block after transcatheter closure of ventricular septal defect |
title_full | Cardiac resynchronization therapy for heart failure induced by left bundle branch block after transcatheter closure of ventricular septal defect |
title_fullStr | Cardiac resynchronization therapy for heart failure induced by left bundle branch block after transcatheter closure of ventricular septal defect |
title_full_unstemmed | Cardiac resynchronization therapy for heart failure induced by left bundle branch block after transcatheter closure of ventricular septal defect |
title_short | Cardiac resynchronization therapy for heart failure induced by left bundle branch block after transcatheter closure of ventricular septal defect |
title_sort | cardiac resynchronization therapy for heart failure induced by left bundle branch block after transcatheter closure of ventricular septal defect |
topic | Case Report |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4294153/ https://www.ncbi.nlm.nih.gov/pubmed/25593586 http://dx.doi.org/10.11909/j.issn.1671-5411.2014.04.012 |
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