Cargando…
Intracellular fibril formation, calcification, and enrichment of chaperones, cytoskeletal, and intermediate filament proteins in the adult hippocampus CA1 following neonatal exposure to the nonprotein amino acid BMAA
The environmental neurotoxin β-N-methylamino-l-alanine (BMAA) has been implicated in the etiology of neurodegenerative disease, and recent studies indicate that BMAA can be misincorporated into proteins. BMAA is a developmental neurotoxicant that can induce long-term learning and memory deficits, as...
Autores principales: | Karlsson, Oskar, Berg, Anna-Lena, Hanrieder, Jörg, Arnerup, Gunnel, Lindström, Anna-Karin, Brittebo, Eva B. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Springer Berlin Heidelberg
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4335130/ https://www.ncbi.nlm.nih.gov/pubmed/24798087 http://dx.doi.org/10.1007/s00204-014-1262-2 |
Ejemplares similares
-
The cyanobacterial neurotoxin β-N-methylamino-l-alanine (BMAA) targets the olfactory bulb region
por: Pierozan, Paula, et al.
Publicado: (2020) -
Neonatal Exposure to the Cyanobacterial Toxin BMAA Induces Changes in Protein Expression and Neurodegeneration in Adult Hippocampus
por: Karlsson, Oskar, et al.
Publicado: (2012) -
Impairment of bile acid metabolism by perfluorooctanoic acid (PFOA) and perfluorooctanesulfonic acid (PFOS) in human HepaRG hepatoma cells
por: Behr, Anne-Cathrin, et al.
Publicado: (2020) -
Human neuronal signaling and communication assays to assess functional neurotoxicity
por: Loser, Dominik, et al.
Publicado: (2020) -
Maternal Transfer of the Cyanobacterial Neurotoxin β-N-Methylamino-L-Alanine (BMAA) via Milk to Suckling Offspring
por: Andersson, Marie, et al.
Publicado: (2013)