Cargando…
Exposure to Diesel Exhaust Particle Extracts (DEPe) Impairs Some Polarization Markers and Functions of Human Macrophages through Activation of AhR and Nrf2
Macrophages (MΦ), well-known to play an important role in immune response, also respond to environmental toxic chemicals such as diesel exhaust particles (DEP). Potential effects of DEPs towards MΦ polarization, a key hall-mark of MΦ physiology, remain however poorly documented. This study was there...
Autores principales: | Jaguin, Marie, Fardel, Olivier, Lecureur, Valérie |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4339390/ https://www.ncbi.nlm.nih.gov/pubmed/25710172 http://dx.doi.org/10.1371/journal.pone.0116560 |
Ejemplares similares
-
Regulation of Human Hepatic Drug Transporter Activity and Expression by Diesel Exhaust Particle Extract
por: Le Vee, Marc, et al.
Publicado: (2015) -
Lipophilic components of diesel exhaust particles induce pro-inflammatory responses in human endothelial cells through AhR dependent pathway(s)
por: Brinchmann, Bendik C., et al.
Publicado: (2018) -
The Landscape of AhR Regulators and Coregulators to Fine-Tune AhR Functions
por: Gargaro, Marco, et al.
Publicado: (2021) -
Investigation of Nrf2, AhR and ATF4 Activation in Toxicogenomic Databases
por: Zgheib, Elias, et al.
Publicado: (2018) -
AhR activation attenuates calcium oxalate nephrocalcinosis by diminishing M1 macrophage polarization and promoting M2 macrophage polarization
por: Yang, Xiaoqi, et al.
Publicado: (2020)