Signaling pathway activation drift during aging: Hutchinson-Gilford Progeria Syndrome fibroblasts are comparable to normal middle-age and old-age cells

For the past several decades, research in understanding the molecular basis of human aging has progressed significantly with the analysis of premature aging syndromes. Progerin, an altered form of lamin A, has been identified as the cause of premature aging in Hutchinson-Gilford Progeria Syndrome (H...

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Detalles Bibliográficos
Autores principales: Aliper, Alexander M., Csoka, Antonei Benjamin, Buzdin, Anton, Jetka, Tomasz, Roumiantsev, Sergey, Moskalev, Alexey, Zhavoronkov, Alex
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals LLC 2015
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4350323/
https://www.ncbi.nlm.nih.gov/pubmed/25587796

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4350323/
https://www.ncbi.nlm.nih.gov/pubmed/25587796

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