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Overexpression of N-terminal kinase like gene promotes tumorigenicity of hepatocellular carcinoma by regulating cell cycle progression and cell motility

Amplification and overexpression of CHD1L is one of the most frequent genetic alterations in hepatocellular carcinoma (HCC). Here we found that one of CHD1L downstream targets, NTKL, was frequently upregulated in HCC, which was significantly correlated with vascular invasion (P = 0.012) and poor pro...

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Detalles Bibliográficos
Autores principales: Wang, Jian, Liu, Ming, Chen, Leilei, Chan, Tim Hon Man, Jiang, Lingxi, Yuan, Yun-Fei, Guan, Xin-Yuan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals LLC 2015
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4359319/
https://www.ncbi.nlm.nih.gov/pubmed/25575811