The intellectual disability protein RAB39B selectively regulates GluA2 trafficking to determine synaptic AMPAR composition

RAB39B is a member of the RAB family of small GTPases that controls intracellular vesicular trafficking in a compartment-specific manner. Mutations in the RAB39B gene cause intellectual disability comorbid with autism spectrum disorder and epilepsy, but the impact of RAB39B loss of function on synap...

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Detalles Bibliográficos
Autores principales: Mignogna, Maria Lidia, Giannandrea, Maila, Gurgone, Antonia, Fanelli, Francesca, Raimondi, Francesco, Mapelli, Lisa, Bassani, Silvia, Fang, Huaqiang, Van Anken, Eelco, Alessio, Massimo, Passafaro, Maria, Gatti, Silvia, Esteban, José A., Huganir, Richard, D’Adamo, Patrizia
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Pub. Group 2015
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4383008/
https://www.ncbi.nlm.nih.gov/pubmed/25784538
http://dx.doi.org/10.1038/ncomms7504

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4383008/
https://www.ncbi.nlm.nih.gov/pubmed/25784538
http://dx.doi.org/10.1038/ncomms7504

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