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Reactive Oxygen Species, Apoptosis, and Mitochondrial Dysfunction in Hearing Loss

Reactive oxygen species (ROS) production is involved in several apoptotic and necrotic cell death pathways in auditory tissues. These pathways are the major causes of most types of sensorineural hearing loss, including age-related hearing loss, hereditary hearing loss, ototoxic drug-induced hearing...

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Detalles Bibliográficos
Autores principales: Kamogashira, Teru, Fujimoto, Chisato, Yamasoba, Tatsuya
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi Publishing Corporation 2015
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4385658/
https://www.ncbi.nlm.nih.gov/pubmed/25874222
http://dx.doi.org/10.1155/2015/617207
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author Kamogashira, Teru
Fujimoto, Chisato
Yamasoba, Tatsuya
author_facet Kamogashira, Teru
Fujimoto, Chisato
Yamasoba, Tatsuya
author_sort Kamogashira, Teru
collection PubMed
description Reactive oxygen species (ROS) production is involved in several apoptotic and necrotic cell death pathways in auditory tissues. These pathways are the major causes of most types of sensorineural hearing loss, including age-related hearing loss, hereditary hearing loss, ototoxic drug-induced hearing loss, and noise-induced hearing loss. ROS production can be triggered by dysfunctional mitochondrial oxidative phosphorylation and increases or decreases in ROS-related enzymes. Although apoptotic cell death pathways are mostly activated by ROS production, there are other pathways involved in hearing loss that do not depend on ROS production. Further studies of other pathways, such as endoplasmic reticulum stress and necrotic cell death, are required.
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spelling pubmed-43856582015-04-13 Reactive Oxygen Species, Apoptosis, and Mitochondrial Dysfunction in Hearing Loss Kamogashira, Teru Fujimoto, Chisato Yamasoba, Tatsuya Biomed Res Int Review Article Reactive oxygen species (ROS) production is involved in several apoptotic and necrotic cell death pathways in auditory tissues. These pathways are the major causes of most types of sensorineural hearing loss, including age-related hearing loss, hereditary hearing loss, ototoxic drug-induced hearing loss, and noise-induced hearing loss. ROS production can be triggered by dysfunctional mitochondrial oxidative phosphorylation and increases or decreases in ROS-related enzymes. Although apoptotic cell death pathways are mostly activated by ROS production, there are other pathways involved in hearing loss that do not depend on ROS production. Further studies of other pathways, such as endoplasmic reticulum stress and necrotic cell death, are required. Hindawi Publishing Corporation 2015 2015-03-22 /pmc/articles/PMC4385658/ /pubmed/25874222 http://dx.doi.org/10.1155/2015/617207 Text en Copyright © 2015 Teru Kamogashira et al. https://creativecommons.org/licenses/by/3.0/ This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Review Article
Kamogashira, Teru
Fujimoto, Chisato
Yamasoba, Tatsuya
Reactive Oxygen Species, Apoptosis, and Mitochondrial Dysfunction in Hearing Loss
title Reactive Oxygen Species, Apoptosis, and Mitochondrial Dysfunction in Hearing Loss
title_full Reactive Oxygen Species, Apoptosis, and Mitochondrial Dysfunction in Hearing Loss
title_fullStr Reactive Oxygen Species, Apoptosis, and Mitochondrial Dysfunction in Hearing Loss
title_full_unstemmed Reactive Oxygen Species, Apoptosis, and Mitochondrial Dysfunction in Hearing Loss
title_short Reactive Oxygen Species, Apoptosis, and Mitochondrial Dysfunction in Hearing Loss
title_sort reactive oxygen species, apoptosis, and mitochondrial dysfunction in hearing loss
topic Review Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4385658/
https://www.ncbi.nlm.nih.gov/pubmed/25874222
http://dx.doi.org/10.1155/2015/617207
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