Cargando…
IκBβ enhances the generation of the low-affinity NFκB/RelA homodimer
The NFκB family of dimeric transcription factors regulate inflammatory and immune responses. While the dynamic control of NFκB dimer activity via the IκB-NFκB signaling module is well understood, there is little information on how specific dimer repertoires are generated from Rel family polypeptides...
Autores principales: | Tsui, Rachel, Kearns, Jeffrey D., Lynch, Candace, Vu, Don, Ngo, Kim, Basak, Soumen, Ghosh, Gourisankar, Hoffmann, Alexander |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4425231/ https://www.ncbi.nlm.nih.gov/pubmed/25946967 http://dx.doi.org/10.1038/ncomms8068 |
Ejemplares similares
-
Dissecting the Regulatory Strategies of NF-κB RelA Target Genes in the Inflammatory Response Reveals Differential Transactivation Logics
por: Ngo, Kim A., et al.
Publicado: (2020) -
NF-κB/Rel Transcription Factors in Pancreatic Cancer: Focusing on RelA, c-Rel, and RelB
por: Kabacaoglu, Derya, et al.
Publicado: (2019) -
NF-κB Repression by PIAS3 Mediated RelA SUMOylation
por: Liu, Yuangang, et al.
Publicado: (2012) -
Regulation of NF-κB activity by competition between RelA acetylation and ubiquitination
por: Li, Haiying, et al.
Publicado: (2011) -
Requirement for the NF-κB Family Member RelA in the Development of Secondary Lymphoid Organs
por: Alcamo, Elizabeth, et al.
Publicado: (2002)