Cargando…
High-Risk Human Papillomavirus Targets Crossroads in Immune Signaling
Persistent infections with a high-risk type human papillomavirus (hrHPV) can progress to cancer. High-risk HPVs infect keratinocytes (KCs) and successfully suppress host immunity for up to two years despite the fact that KCs are well equipped to detect and initiate immune responses to invading patho...
Autores principales: | Tummers, Bart, Van Der Burg, Sjoerd H. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4452916/ https://www.ncbi.nlm.nih.gov/pubmed/26008697 http://dx.doi.org/10.3390/v7052485 |
Ejemplares similares
-
Human Papillomavirus (HPV) Upregulates the Cellular Deubiquitinase UCHL1 to Suppress the Keratinocyte's Innate Immune Response
por: Karim, Rezaul, et al.
Publicado: (2013) -
Targeting KRAS: Crossroads of Signaling and Immune Inhibition
por: Kato, Shumei, et al.
Publicado: (2022) -
Human Papillomavirus Downregulates the Expression of IFITM1 and RIPK3 to Escape from IFNγ- and TNFα-Mediated Antiproliferative Effects and Necroptosis
por: Ma, Wenbo, et al.
Publicado: (2016) -
The interferon-related developmental regulator 1 is used by human papillomavirus to suppress NFκB activation
por: Tummers, Bart, et al.
Publicado: (2015) -
CD40-Mediated Amplification of Local Immunity by Epithelial Cells Is Impaired by HPV
por: Tummers, Bart, et al.
Publicado: (2014)