Cargando…
Oxidative stress, mitochondrial damage, and cores in muscle from calsequestrin-1 knockout mice
BACKGROUND: Mutations in the gene encoding ryanodine receptor type-1 (RYR1), the calcium ion (Ca(2+)) release channel in the sarcoplasmic reticulum (SR) of skeletal muscle, are linked to central core disease (CCD) and malignant hyperthermia (MH) susceptibility. We recently reported that mice lacking...
Autores principales: | Paolini, Cecilia, Quarta, Marco, Wei-LaPierre, Lan, Michelucci, Antonio, Nori, Alessandra, Reggiani, Carlo, Dirksen, Robert T, Protasi, Feliciano |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4464246/ https://www.ncbi.nlm.nih.gov/pubmed/26075051 http://dx.doi.org/10.1186/s13395-015-0035-9 |
Ejemplares similares
-
Differential Effect of Calsequestrin Ablation on Structure and Function of Fast and Slow Skeletal Muscle Fibers
por: Paolini, Cecilia, et al.
Publicado: (2011) -
Estrogens Protect Calsequestrin-1 Knockout Mice from Lethal Hyperthermic Episodes by Reducing Oxidative Stress in Muscle
por: Michelucci, Antonio, et al.
Publicado: (2017) -
Constitutive assembly of Ca(2+) entry units in soleus muscle from calsequestrin knockout mice
por: Michelucci, Antonio, et al.
Publicado: (2022) -
Pre-assembled Ca(2+) entry units and constitutively active Ca(2+) entry in skeletal muscle of calsequestrin-1 knockout mice
por: Michelucci, Antonio, et al.
Publicado: (2020) -
Mitochondrial Ca(2+)-Handling in Fast Skeletal Muscle Fibers from Wild Type and Calsequestrin-Null Mice
por: Scorzeto, Michele, et al.
Publicado: (2013)