Cargando…
The Na(v) channel bench series: Plasmid preparation()
Research involving recombinant voltage-gated sodium (Na(v)) channels has unique challenges. Multiple factors contribute, but undoubtedly at the top of the list is these channels’ DNA instability. Once introduced into bacterial hosts, Na(v) channel plasmid DNA will almost invariably emerge mutagenize...
Autores principales: | Feldman, Daniel H., Lossin, Christoph |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4472833/ https://www.ncbi.nlm.nih.gov/pubmed/26150927 http://dx.doi.org/10.1016/j.mex.2014.01.002 |
Ejemplares similares
-
Single-channel Properties of Human Na(V)1.1 and Mechanism of Channel Dysfunction in SCN1A-associated Epilepsy
por: Vanoye, Carlos G., et al.
Publicado: (2006) -
Acidosis Differentially Modulates Inactivation in Na(V)1.2, Na(V)1.4, and Na(V)1.5 Channels
por: Vilin, Yury Y., et al.
Publicado: (2012) -
From Bench to Biomolecular Simulation: Phospholipid Modulation of Potassium Channels
por: Pipatpolkai, Tanadet, et al.
Publicado: (2021) -
Na(V)1.6 and Na(V)1.7 channels are major endogenous voltage-gated sodium channels in ND7/23 cells
por: Lee, Jisoo, et al.
Publicado: (2019) -
Novel HCN2 Mutation Contributes to Febrile Seizures by Shifting the Channel's Kinetics in a Temperature-Dependent Manner
por: Nakamura, Yuki, et al.
Publicado: (2013)