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Increased Mitochondrial Activity in Anthrax-Induced Cell Death

Pathogenesis of anthrax lethal toxin (LT) is attributed to its ability to cause death of infected cells. New work has demonstrated that increase of mitochondrial F(1)F(0) ATPase activity and subsequent depletion of cellular ATP level are critical early events during LT-induced cell death.

Detalles Bibliográficos
Autor principal: Li, Chi
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Libertas Academica 2009
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4474338/
https://www.ncbi.nlm.nih.gov/pubmed/26124679

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