Regulation of NEIL1 protein abundance by RAD9 is important for efficient base excision repair

RAD9 participates in DNA damage-induced cell cycle checkpoints and DNA repair. As a member of the RAD9-HUS1-RAD1 (9-1-1) complex, it can sense DNA damage and recruit ATR to damage sites. RAD9 binding can enhance activities of members of different DNA repair pathways, including NEIL1 DNA glycosylase,...

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Detalles Bibliográficos
Autores principales: Panigrahi, Sunil K., Hopkins, Kevin M., Lieberman, Howard B.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Oxford University Press 2015
Materias:
Genome Integrity, Repair and Replication
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4482081/
https://www.ncbi.nlm.nih.gov/pubmed/25873625
http://dx.doi.org/10.1093/nar/gkv327

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4482081/
https://www.ncbi.nlm.nih.gov/pubmed/25873625
http://dx.doi.org/10.1093/nar/gkv327

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