Cargando…
Sesamin Ameliorates Advanced Glycation End Products-Induced Pancreatic β-Cell Dysfunction and Apoptosis
Advanced glycation end products (AGEs), the direct modulators of β-cells, have been shown to cause insulin-producing β-cell dysfunction and apoptosis through increase of intracellular reactive oxygen species (ROS) production. Sesamin has been demonstrated to possess antioxidative activity. This stud...
Autores principales: | Kong, Xiang, Wang, Guo-Dong, Ma, Ming-Zhe, Deng, Ru-Yuan, Guo, Li-Qun, Zhang, Jun-Xiu, Yang, Jie-Ren, Su, Qing |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
MDPI
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4488808/ https://www.ncbi.nlm.nih.gov/pubmed/26066015 http://dx.doi.org/10.3390/nu7064689 |
Ejemplares similares
-
Differentiation therapy: sesamin as an effective agent in targeting cancer stem-like side population cells of human gallbladder carcinoma
por: Kong, Xiang, et al.
Publicado: (2014) -
Sesamin: A Promising Therapeutic Agent for Ameliorating Symptoms of Diabetes
por: Huang, Shu-Ming, et al.
Publicado: (2023) -
Effects of Sesamin on Streptozotocin (STZ)-Induced NIT-1 Pancreatic β-Cell Damage
por: Lei, Hong, et al.
Publicado: (2012) -
Novel therapeutic effects of sesamin on diabetes-induced cardiac dysfunction
por: Thuy, Tran Duong, et al.
Publicado: (2017) -
Sesamin Ameliorates High-Fat Diet–Induced Dyslipidemia and Kidney Injury by Reducing Oxidative Stress
por: Zhang, Ruijuan, et al.
Publicado: (2016)