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SNAREs support atlastin-mediated homotypic ER fusion in Saccharomyces cerevisiae
Dynamin-like GTPases of the atlastin family are thought to mediate homotypic endoplasmic reticulum (ER) membrane fusion; however, the underlying mechanism remains largely unclear. Here, we developed a simple and quantitative in vitro assay using isolated yeast microsomes for measuring yeast atlastin...
Autores principales: | , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
2015
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4523606/ https://www.ncbi.nlm.nih.gov/pubmed/26216899 http://dx.doi.org/10.1083/jcb.201501043 |
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author | Lee, Miriam Ko, Young-Joon Moon, Yeojin Han, Minsoo Kim, Hyung-Wook Lee, Sung Haeng Kang, KyeongJin Jun, Youngsoo |
author_facet | Lee, Miriam Ko, Young-Joon Moon, Yeojin Han, Minsoo Kim, Hyung-Wook Lee, Sung Haeng Kang, KyeongJin Jun, Youngsoo |
author_sort | Lee, Miriam |
collection | PubMed |
description | Dynamin-like GTPases of the atlastin family are thought to mediate homotypic endoplasmic reticulum (ER) membrane fusion; however, the underlying mechanism remains largely unclear. Here, we developed a simple and quantitative in vitro assay using isolated yeast microsomes for measuring yeast atlastin Sey1p-dependent ER fusion. Using this assay, we found that the ER SNAREs Sec22p and Sec20p were required for Sey1p-mediated ER fusion. Consistently, ER fusion was significantly reduced by inhibition of Sec18p and Sec17p, which regulate SNARE-mediated membrane fusion. The involvement of SNAREs in Sey1p-dependent ER fusion was further supported by the physical interaction of Sey1p with Sec22p and Ufe1p, another ER SNARE. Furthermore, our estimation of the concentration of Sey1p on isolated microsomes, together with the lack of fusion between Sey1p proteoliposomes even with a 25-fold excess of the physiological concentration of Sey1p, suggests that Sey1p requires additional factors to support ER fusion in vivo. Collectively, our data strongly suggest that SNARE-mediated membrane fusion is involved in atlastin-initiated homotypic ER fusion. |
format | Online Article Text |
id | pubmed-4523606 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2015 |
publisher | The Rockefeller University Press |
record_format | MEDLINE/PubMed |
spelling | pubmed-45236062016-02-03 SNAREs support atlastin-mediated homotypic ER fusion in Saccharomyces cerevisiae Lee, Miriam Ko, Young-Joon Moon, Yeojin Han, Minsoo Kim, Hyung-Wook Lee, Sung Haeng Kang, KyeongJin Jun, Youngsoo J Cell Biol Research Articles Dynamin-like GTPases of the atlastin family are thought to mediate homotypic endoplasmic reticulum (ER) membrane fusion; however, the underlying mechanism remains largely unclear. Here, we developed a simple and quantitative in vitro assay using isolated yeast microsomes for measuring yeast atlastin Sey1p-dependent ER fusion. Using this assay, we found that the ER SNAREs Sec22p and Sec20p were required for Sey1p-mediated ER fusion. Consistently, ER fusion was significantly reduced by inhibition of Sec18p and Sec17p, which regulate SNARE-mediated membrane fusion. The involvement of SNAREs in Sey1p-dependent ER fusion was further supported by the physical interaction of Sey1p with Sec22p and Ufe1p, another ER SNARE. Furthermore, our estimation of the concentration of Sey1p on isolated microsomes, together with the lack of fusion between Sey1p proteoliposomes even with a 25-fold excess of the physiological concentration of Sey1p, suggests that Sey1p requires additional factors to support ER fusion in vivo. Collectively, our data strongly suggest that SNARE-mediated membrane fusion is involved in atlastin-initiated homotypic ER fusion. The Rockefeller University Press 2015-08-03 /pmc/articles/PMC4523606/ /pubmed/26216899 http://dx.doi.org/10.1083/jcb.201501043 Text en © 2015 Lee et al. This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/). |
spellingShingle | Research Articles Lee, Miriam Ko, Young-Joon Moon, Yeojin Han, Minsoo Kim, Hyung-Wook Lee, Sung Haeng Kang, KyeongJin Jun, Youngsoo SNAREs support atlastin-mediated homotypic ER fusion in Saccharomyces cerevisiae |
title | SNAREs support atlastin-mediated homotypic ER fusion in Saccharomyces cerevisiae |
title_full | SNAREs support atlastin-mediated homotypic ER fusion in Saccharomyces cerevisiae |
title_fullStr | SNAREs support atlastin-mediated homotypic ER fusion in Saccharomyces cerevisiae |
title_full_unstemmed | SNAREs support atlastin-mediated homotypic ER fusion in Saccharomyces cerevisiae |
title_short | SNAREs support atlastin-mediated homotypic ER fusion in Saccharomyces cerevisiae |
title_sort | snares support atlastin-mediated homotypic er fusion in saccharomyces cerevisiae |
topic | Research Articles |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4523606/ https://www.ncbi.nlm.nih.gov/pubmed/26216899 http://dx.doi.org/10.1083/jcb.201501043 |
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