Cargando…
Therapeutic effects of intravenous administration of bone marrow stromal cells on sevoflurane-induced neuronal apoptosis and neuroinflammation in neonatal rats
BACKGROUND: Sevoflurane exposure during the early postnatal period causes neuroinflammation and neuronal apoptosis in rodents. Bone marrow stromal cells (BMSCs) have been shown to protect and repair the damaged central nervous system, for example in ischemic stroke models. In this study, we investig...
Autores principales: | Sun, ZhongLiang, Satomoto, Maiko, Makita, Koshi |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
The Korean Society of Anesthesiologists
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4524940/ https://www.ncbi.nlm.nih.gov/pubmed/26257854 http://dx.doi.org/10.4097/kjae.2015.68.4.397 |
Ejemplares similares
-
Sugammadex-Enhanced Neuronal Apoptosis following Neonatal Sevoflurane Exposure in Mice
por: Satomoto, Maiko, et al.
Publicado: (2016) -
Corrigendum to “Sugammadex-Enhanced Neuronal Apoptosis following Neonatal Sevoflurane Exposure in Mice”
por: Satomoto, Maiko, et al.
Publicado: (2017) -
Apocynin preserves glutamatergic neurons in the basolateral amygdala in mice with neonatal sevoflurane exposure
por: Sun, Zhongliang, et al.
Publicado: (2017) -
Blood–brain barrier disruption caused by neonatal sevoflurane-induced depends on exposure time and is reversible in mice
por: Sun, Zhongliang, et al.
Publicado: (2019) -
Sevoflurane preconditioning ameliorates lipopolysaccharide-induced cognitive
impairment in mice
por: Satomoto, Maiko, et al.
Publicado: (2017)