Cargando…
DNA Damage Response Checkpoint Activation Drives KP1019 Dependent Pre-Anaphase Cell Cycle Delay in S. cerevisiae
Careful regulation of the cell cycle is required for proper replication, cell division, and DNA repair. DNA damage–including that induced by many anticancer drugs–results in cell cycle delay or arrest, which can allow time for repair of DNA lesions. Although its molecular mechanism of action remains...
Autores principales: | Bierle, Lindsey A., Reich, Kira L., Taylor, Braden E., Blatt, Eliot B., Middleton, Sydney M., Burke, Shawnecca D., Stultz, Laura K., Hanson, Pamela K., Partridge, Janet F., Miller, Mary E. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4572706/ https://www.ncbi.nlm.nih.gov/pubmed/26375390 http://dx.doi.org/10.1371/journal.pone.0138085 |
Ejemplares similares
-
Correction: DNA Damage Response Checkpoint Activation Drives KP1019 Dependent Pre-Anaphase Cell Cycle Delay in S. cerevisiae
Publicado: (2015) -
DMSO reduces the cytotoxicity of anticancer ruthenium complex KP1019 in yeast
por: Davis, Jonathan, et al.
Publicado: (2021) -
Inhibitory Effects of the Ruthenium Complex KP1019 in Models of Mammary Cancer Cell Migration and Invasion
por: Bergamo, A., et al.
Publicado: (2009) -
A systematic assessment of chemical, genetic, and epigenetic factors influencing the activity of anticancer drug KP1019 (FFC14A)
por: Golla, Upendarrao, et al.
Publicado: (2017) -
En Route to Osmium Analogues of KP1019: Synthesis, Structure, Spectroscopic Properties and Antiproliferative Activity of trans-[Os(IV)Cl(4)(Hazole)(2)]
por: Büchel, Gabriel E., et al.
Publicado: (2011)