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Impaired PIEZO1 function in patients with a novel autosomal recessive congenital lymphatic dysplasia

Piezo1 ion channels are mediators of mechanotransduction in several cell types including the vascular endothelium, renal tubular cells and erythrocytes. Gain-of-function mutations in PIEZO1 cause an autosomal dominant haemolytic anaemia in humans called dehydrated hereditary stomatocytosis. However,...

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Autores principales: Lukacs, Viktor, Mathur, Jayanti, Mao, Rong, Bayrak-Toydemir, Pinar, Procter, Melinda, Cahalan, Stuart M., Kim, Helen J., Bandell, Michael, Longo, Nicola, Day, Ronald W., Stevenson, David A., Patapoutian, Ardem, Krock, Bryan L.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2015
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4578306/
https://www.ncbi.nlm.nih.gov/pubmed/26387913
http://dx.doi.org/10.1038/ncomms9329
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author Lukacs, Viktor
Mathur, Jayanti
Mao, Rong
Bayrak-Toydemir, Pinar
Procter, Melinda
Cahalan, Stuart M.
Kim, Helen J.
Bandell, Michael
Longo, Nicola
Day, Ronald W.
Stevenson, David A.
Patapoutian, Ardem
Krock, Bryan L.
author_facet Lukacs, Viktor
Mathur, Jayanti
Mao, Rong
Bayrak-Toydemir, Pinar
Procter, Melinda
Cahalan, Stuart M.
Kim, Helen J.
Bandell, Michael
Longo, Nicola
Day, Ronald W.
Stevenson, David A.
Patapoutian, Ardem
Krock, Bryan L.
author_sort Lukacs, Viktor
collection PubMed
description Piezo1 ion channels are mediators of mechanotransduction in several cell types including the vascular endothelium, renal tubular cells and erythrocytes. Gain-of-function mutations in PIEZO1 cause an autosomal dominant haemolytic anaemia in humans called dehydrated hereditary stomatocytosis. However, the phenotypic consequence of PIEZO1 loss of function in humans has not previously been documented. Here we discover a novel role of this channel in the lymphatic system. Through whole-exome sequencing, we identify biallelic mutations in PIEZO1 (a splicing variant leading to early truncation and a non-synonymous missense variant) in a pair of siblings affected with persistent lymphoedema caused by congenital lymphatic dysplasia. Analysis of patients' erythrocytes as well as studies in a heterologous system reveal greatly attenuated PIEZO1 function in affected alleles. Our results delineate a novel clinical category of PIEZO1-associated hereditary lymphoedema.
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spelling pubmed-45783062015-10-21 Impaired PIEZO1 function in patients with a novel autosomal recessive congenital lymphatic dysplasia Lukacs, Viktor Mathur, Jayanti Mao, Rong Bayrak-Toydemir, Pinar Procter, Melinda Cahalan, Stuart M. Kim, Helen J. Bandell, Michael Longo, Nicola Day, Ronald W. Stevenson, David A. Patapoutian, Ardem Krock, Bryan L. Nat Commun Article Piezo1 ion channels are mediators of mechanotransduction in several cell types including the vascular endothelium, renal tubular cells and erythrocytes. Gain-of-function mutations in PIEZO1 cause an autosomal dominant haemolytic anaemia in humans called dehydrated hereditary stomatocytosis. However, the phenotypic consequence of PIEZO1 loss of function in humans has not previously been documented. Here we discover a novel role of this channel in the lymphatic system. Through whole-exome sequencing, we identify biallelic mutations in PIEZO1 (a splicing variant leading to early truncation and a non-synonymous missense variant) in a pair of siblings affected with persistent lymphoedema caused by congenital lymphatic dysplasia. Analysis of patients' erythrocytes as well as studies in a heterologous system reveal greatly attenuated PIEZO1 function in affected alleles. Our results delineate a novel clinical category of PIEZO1-associated hereditary lymphoedema. Nature Publishing Group 2015-09-21 /pmc/articles/PMC4578306/ /pubmed/26387913 http://dx.doi.org/10.1038/ncomms9329 Text en Copyright © 2015, Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. http://creativecommons.org/licenses/by/4.0/ This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/
spellingShingle Article
Lukacs, Viktor
Mathur, Jayanti
Mao, Rong
Bayrak-Toydemir, Pinar
Procter, Melinda
Cahalan, Stuart M.
Kim, Helen J.
Bandell, Michael
Longo, Nicola
Day, Ronald W.
Stevenson, David A.
Patapoutian, Ardem
Krock, Bryan L.
Impaired PIEZO1 function in patients with a novel autosomal recessive congenital lymphatic dysplasia
title Impaired PIEZO1 function in patients with a novel autosomal recessive congenital lymphatic dysplasia
title_full Impaired PIEZO1 function in patients with a novel autosomal recessive congenital lymphatic dysplasia
title_fullStr Impaired PIEZO1 function in patients with a novel autosomal recessive congenital lymphatic dysplasia
title_full_unstemmed Impaired PIEZO1 function in patients with a novel autosomal recessive congenital lymphatic dysplasia
title_short Impaired PIEZO1 function in patients with a novel autosomal recessive congenital lymphatic dysplasia
title_sort impaired piezo1 function in patients with a novel autosomal recessive congenital lymphatic dysplasia
topic Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4578306/
https://www.ncbi.nlm.nih.gov/pubmed/26387913
http://dx.doi.org/10.1038/ncomms9329
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