Cargando…
Investigation of bifurcation properties of a model of myotonia caused by incomplete inactivation of sodium channels in skeletal muscle fibers
Autores principales: | Kocharoen, Kamonwan, Bell, Jonathan |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2007
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4578827/ http://dx.doi.org/10.1186/1471-2202-8-S2-P125 |
Ejemplares similares
-
Isolated eyelid closure myotonia in two families with sodium channel myotonia
por: Stunnenberg, B. C., et al.
Publicado: (2009) -
Inactivation defects caused by myotonia-associated mutations in the sodium channel III-IV linker
Publicado: (1996) -
Slow sodium-channel inactivation underlies spike threshold variability
por: Harrison, Paul M, et al.
Publicado: (2013) -
A Mixed Periodic Paralysis & Myotonia Mutant, P1158S, Imparts pH-Sensitivity in Skeletal Muscle Voltage-gated Sodium Channels
por: Ghovanloo, Mohammad-Reza, et al.
Publicado: (2018) -
Interactions of neosaxitoxin with the sodium channel of the frog skeletal muscle fiber
Publicado: (1991)