Cargando…
Systemic and Cardiac Depletion of M2 Macrophage through CSF-1R Signaling Inhibition Alters Cardiac Function Post Myocardial Infarction
The heart hosts tissue resident macrophages which are capable of modulating cardiac inflammation and function by multiple mechanisms. At present, the consequences of phenotypic diversity in macrophages in the heart are incompletely understood. The contribution of cardiac M2-polarized macrophages to...
Autores principales: | Leblond, Anne-Laure, Klinkert, Kerstin, Martin, Kenneth, Turner, Elizebeth C., Kumar, Arun H., Browne, Tara, Caplice, Noel M. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4583226/ https://www.ncbi.nlm.nih.gov/pubmed/26407006 http://dx.doi.org/10.1371/journal.pone.0137515 |
Ejemplares similares
-
Bone marrow mononuclear stem cells: potential in the treatment of myocardial infarction
por: Leblond, Anne-Laure, et al.
Publicado: (2009) -
Role of CX(3)CR1 Receptor in Monocyte/Macrophage Driven Neovascularization
por: Kumar, Arun H. S., et al.
Publicado: (2013) -
Cardiac monocytes and macrophages after myocardial infarction
por: Peet, Claire, et al.
Publicado: (2020) -
Apoptosis inhibitor of macrophage depletion decreased M1 macrophage accumulation and the incidence of cardiac rupture after myocardial infarction in mice
por: Ishikawa, Shohei, et al.
Publicado: (2017) -
Combinatorial G-CSF/AMD3100 Treatment in Cardiac Repair after Myocardial Infarction
por: Rüder, Constantin, et al.
Publicado: (2014)