Cargando…

Environmental enrichment does not influence hypersynchronous network activity in the Tg2576 mouse model of Alzheimer’s disease

The cognitive reserve hypothesis claims that the brain can overcome pathology by reinforcing preexistent processes or by developing alternative cognitive strategies. Epidemiological studies have revealed that this reserve can be built throughout life experiences as education or leisure activities. W...

Descripción completa

Detalles Bibliográficos
Autores principales:	Bezzina, Charlotte, Verret, Laure, Halley, Hélène, Dahan, Lionel, Rampon, Claire
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	Frontiers Media S.A. 2015
Materias:	Neuroscience
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4585132/ https://www.ncbi.nlm.nih.gov/pubmed/26441640 http://dx.doi.org/10.3389/fnagi.2015.00178

Ejemplares similares

Early Onset of Hypersynchronous Network Activity and Expression of a Marker of Chronic Seizures in the Tg2576 Mouse Model of Alzheimer’s Disease
por: Bezzina, Charlotte, et al.
Publicado: (2015)

Modifications of Hippocampal Circuits and Early Disruption of Adult Neurogenesis in the Tg2576 Mouse Model of Alzheimer’s Disease
por: Krezymon, Alice, et al.
Publicado: (2013)

Trehalose Improves Cognition in the Transgenic Tg2576 Mouse Model of Alzheimer’s Disease
por: Portbury, Stuart D., et al.
Publicado: (2017)

Neuritin Attenuates Cognitive Function Impairments in Tg2576 Mouse Model of Alzheimer's Disease
por: Choi, Yoori, et al.
Publicado: (2014)

A Novel Form of Compensation in the Tg2576 Amyloid Mouse Model of Alzheimer’s Disease
por: Somogyi, Attila, et al.
Publicado: (2016)

Iron Chelation Inhibits Osteoclastic Differentiation In Vitro and in Tg2576 Mouse Model of Alzheimer’s Disease
por: Guo, Jun-Peng, et al.
Publicado: (2015)

Decreased Myelinated Fibers in the Hippocampal Dentate Gyrus of the Tg2576 Mouse Model of Alzheimer’s Disease
por: Lu, Wei, et al.
Publicado: (2016)

Quantitative study of the capillaries within the white matter of the Tg2576 mouse model of Alzheimer's disease
por: Zhang, Yi, et al.
Publicado: (2019)

Paraoxonase-1 and -3 Protein Expression in the Brain of the Tg2576 Mouse Model of Alzheimer’s Disease
por: Salazar, Jose Gregorio, et al.
Publicado: (2021)

Dysbiosis of Fecal Microbiota in Tg2576 Mice for Alzheimer’s Disease during Pathological Constipation
por: Kim, Ji-Eun, et al.
Publicado: (2022)

Mitochondrial Superoxide Contributes to Blood Flow and Axonal Transport Deficits in the Tg2576 Mouse Model of Alzheimer's Disease
por: Massaad, Cynthia A., et al.
Publicado: (2010)

Characterization of hippocampal Cajal-Retzius cells during development in a mouse model of Alzheimer's disease (Tg2576)
por: Yu, Dongming, et al.
Publicado: (2014)

Oxidative Stress during the Progression of β-Amyloid Pathology in the Neocortex of the Tg2576 Mouse Model of Alzheimer's Disease
por: Porcellotti, Sara, et al.
Publicado: (2015)

Enriched environmental exposure reduces the onset of action of the serotonin norepinephrin reuptake inhibitor venlafaxine through its effect on parvalbumin interneurons plasticity in mice
por: Coutens, Basile, et al.
Publicado: (2023)

Analysis of differential plaque depositions in the brains of Tg2576 and Tg-APPswe/PS1dE9 transgenic mouse models of Alzheimer disease
por: Kim, Tae-Kyung, et al.
Publicado: (2012)

Age-related changes of protein SUMOylation balance in the AβPP Tg2576 mouse model of Alzheimer's disease
por: Nisticò, Robert, et al.
Publicado: (2014)

Spared Piriform Cortical Single-Unit Odor Processing and Odor Discrimination in the Tg2576 Mouse Model of Alzheimer's Disease
por: Xu, Wenjin, et al.
Publicado: (2014)

Cell Type-Specific Human APP Transgene Expression by Hippocampal Interneurons in the Tg2576 Mouse Model of Alzheimer’s Disease
por: Höfling, Corinna, et al.
Publicado: (2019)

Spatial and latent memory data in PS2Tg2576 alzheimer's disease mouse model after memantine treatment
por: Matsumura, Masahisa, et al.
Publicado: (2021)

Immunohistochemical Demonstration of the pGlu79 α-Synuclein Fragment in Alzheimer’s Disease and Its Tg2576 Mouse Model
por: Bluhm, Alexandra, et al.
Publicado: (2022)

Defined astrocytic expression of human amyloid precursor protein in Tg2576 mouse brain
por: Heiland, Tina, et al.
Publicado: (2018)

NitroSynapsin ameliorates hypersynchronous neural network activity in Alzheimer hiPSC models
por: Ghatak, Swagata, et al.
Publicado: (2021)

Leaky Ryanodine receptors increases Amyloid-beta load and induces memory impairments in Tg2576 mouse model of Alzheimer disease
por: Oulès, Bénédicte, et al.
Publicado: (2013)

Up-regulation of Synaptotagmin IV within amyloid plaque-associated dystrophic neurons in Tg2576 mouse model of Alzheimer’s disease
por: Tratnjek, Larisa, et al.
Publicado: (2013)

Chronic treatment with a smart antioxidative nanoparticle for inhibition of amyloid plaque propagation in Tg2576 mouse model of Alzheimer’s disease
por: Boonruamkaew, Phetcharat, et al.
Publicado: (2017)

ACE2 activation protects against cognitive decline and reduces amyloid pathology in the Tg2576 mouse model of Alzheimer’s disease
por: Evans, Charles E., et al.
Publicado: (2020)

Docosahexaenoic Acid Ameliorates Contextual Fear Memory Deficits in the Tg2576 Alzheimer’s Disease Mouse Model: Cellular and Molecular Correlates
por: Badesso, Sara, et al.
Publicado: (2022)

Age-Dependent Neuroplasticity Mechanisms in Alzheimer Tg2576 Mice Following Modulation of Brain Amyloid-β Levels
por: Lilja, Anna M., et al.
Publicado: (2013)

Deletion of UCP1 in Tg2576 Mice Increases Body Temperature and Exacerbates Alzheimer’s Disease-Related Pathologies
por: Jung, Cha-Gyun, et al.
Publicado: (2023)

Amyloid β-Induced Upregulation of Na(v)1.6 Underlies Neuronal Hyperactivity in Tg2576 Alzheimer’s Disease Mouse Model
por: Ciccone, Roselia, et al.
Publicado: (2019)

Intracerebral Infection with E. coli Impairs Spatial Learning and Induces Necrosis of Hippocampal Neurons in the Tg2576 Mouse Model of Alzheimer’s Disease
por: Schütze, Sandra, et al.
Publicado: (2022)

Correction: Age-Dependent Neuroplasticity Mechanisms in Alzheimer Tg2576 Mice Following Modulation of Brain Amyloid-β Levels
por: Lilja, Anna M., et al.
Publicado: (2013)

Asparagus cochinchinensis stimulates release of nerve growth factor and abrogates oxidative stress in the Tg2576 model for Alzheimer’s disease
por: Lee, Hyun Ah, et al.
Publicado: (2018)

Establishment of an Alzheimer’s disease model with latent herpesvirus infection using PS2 and Tg2576 double transgenic mice
por: Tanaka, Seiichi, et al.
Publicado: (2017)

Interictal spikes during sleep are an early defect in the Tg2576 mouse model of β-amyloid neuropathology
por: Kam, Korey, et al.
Publicado: (2016)

Influence of Transgenic Metallothionein-1 on Gliosis, CA1 Neuronal Loss, and Brain Metal Levels of the Tg2576 Mouse Model of Alzheimer’s Disease
por: Comes, Gemma, et al.
Publicado: (2017)

Local perfusion of capillaries reveals disrupted beta-amyloid homeostasis at the blood-brain barrier in Tg2576 murine Alzheimer’s model
por: Hanafy, Amira Sayed, et al.
Publicado: (2023)

p38 MAP kinase-mediated NMDA receptor-dependent suppression of hippocampal hypersynchronicity in a mouse model of Alzheimer’s disease
por: Ittner, Arne A, et al.
Publicado: (2014)

Exercise Training Results in Lower Amyloid Plaque Load and Greater Cognitive Function in an Intensity Dependent Manner in the Tg2576 Mouse Model of Alzheimer’s Disease
por: Thomas, Riya, et al.
Publicado: (2020)

Ablation of the Locus Coeruleus Increases Oxidative Stress in Tg-2576 Transgenic but Not Wild-Type Mice
por: Hurko, Orest, et al.
Publicado: (2010)

Cannot write session to /tmp/vufind_sessions/sess_rpar3mbmt2g7evb61ba8bf0832