Cargando…
Translation of the focus toward excellence in translational science: comment on “TDP-43 Repression of Nonconserved Cryptic Exons is Compromised in ALS-FTD”
Autores principales: | Pochet, Roland, Nicaise, Charles, Mitrečić, Dinko |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Croatian Medical Schools
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4655934/ https://www.ncbi.nlm.nih.gov/pubmed/26526887 http://dx.doi.org/10.3325/cmj.2015.56.493 |
Ejemplares similares
-
TDP-43 represses cryptic exon inclusion in the FTD–ALS gene UNC13A
por: Ma, X. Rosa, et al.
Publicado: (2022) -
Induction of autophagy mitigates TDP-43 pathology and translational repression of neurofilament mRNAs in mouse models of ALS/FTD
por: Kumar, Sunny, et al.
Publicado: (2021) -
TDP-43 and other hnRNPs regulate cryptic exon inclusion of a key ALS/FTD risk gene, UNC13A
por: Koike, Yuka, et al.
Publicado: (2023) -
Tdp-43 cryptic exons are highly variable between cell types
por: Jeong, Yun Ha, et al.
Publicado: (2017) -
Correction: TDP-43 and other hnRNPs regulate cryptic exon inclusion of a key ALS/FTD risk gene, UNC13A
por: Koike, Yuka, et al.
Publicado: (2023)