Cargando…
Bradykinin inhibits oxidative stress-induced senescence of endothelial progenitor cells through the B2R/AKT/RB and B2R/EGFR/RB signal pathways
Circulating endothelial progenitor cells (EPCs) have multiple protective effects that facilitate repair of damage to tissues and organs. However, while various stressors are known to impair EPC function, the mechanisms of oxidative stress-induced EPC senescence remains unknown. We demonstrated that...
Autores principales: | Fu, Cong, Li, Bing, Sun, Yuning, Ma, Genshan, Yao, Yuyu |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Impact Journals LLC
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4694787/ https://www.ncbi.nlm.nih.gov/pubmed/26360782 |
Ejemplares similares
-
Gadd45b deficiency promotes premature senescence and skin aging
por: Magimaidas, Andrew, et al.
Publicado: (2016) -
Bradykinin Protects Human Endothelial Progenitor Cells from High-Glucose-Induced Senescence through B2 Receptor-Mediated Activation of the Akt/eNOS Signalling Pathway
por: Wu, Yuehuan, et al.
Publicado: (2021) -
MiR-23a-depressed autophagy is a participant in PUVA- and UVB-induced premature senescence
por: Zhang, Jia-an, et al.
Publicado: (2016) -
Association study of polymorphisms in FOXO3, AKT1 and IGF-2R genes with human longevity in a Han Chinese population
por: Li, Ning, et al.
Publicado: (2015) -
The underlying mechanism of proinflammatory NF-κB activation by the mTORC2/Akt/IKKα pathway during skin aging
por: Choi, Yeon Ja, et al.
Publicado: (2016)