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Why Are tRNAs Overproduced in the Absence of Maf1, a Negative Regulator of RNAP III, Not Fully Functional?

Detalles Bibliográficos
Autor principal: Boguta, Magdalena
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2015
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4699899/
https://www.ncbi.nlm.nih.gov/pubmed/26720418
http://dx.doi.org/10.1371/journal.pgen.1005743
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author Boguta, Magdalena
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spelling pubmed-46998992016-01-14 Why Are tRNAs Overproduced in the Absence of Maf1, a Negative Regulator of RNAP III, Not Fully Functional? Boguta, Magdalena PLoS Genet Perspective Public Library of Science 2015-12-31 /pmc/articles/PMC4699899/ /pubmed/26720418 http://dx.doi.org/10.1371/journal.pgen.1005743 Text en © 2015 Magdalena Boguta http://creativecommons.org/licenses/by/4.0/ This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.
spellingShingle Perspective
Boguta, Magdalena
Why Are tRNAs Overproduced in the Absence of Maf1, a Negative Regulator of RNAP III, Not Fully Functional?
title Why Are tRNAs Overproduced in the Absence of Maf1, a Negative Regulator of RNAP III, Not Fully Functional?
title_full Why Are tRNAs Overproduced in the Absence of Maf1, a Negative Regulator of RNAP III, Not Fully Functional?
title_fullStr Why Are tRNAs Overproduced in the Absence of Maf1, a Negative Regulator of RNAP III, Not Fully Functional?
title_full_unstemmed Why Are tRNAs Overproduced in the Absence of Maf1, a Negative Regulator of RNAP III, Not Fully Functional?
title_short Why Are tRNAs Overproduced in the Absence of Maf1, a Negative Regulator of RNAP III, Not Fully Functional?
title_sort why are trnas overproduced in the absence of maf1, a negative regulator of rnap iii, not fully functional?
topic Perspective
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4699899/
https://www.ncbi.nlm.nih.gov/pubmed/26720418
http://dx.doi.org/10.1371/journal.pgen.1005743
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