Cargando…
Ca(2+) signals mediated by bradykinin type 2 receptors in normal pancreatic stellate cells can be inhibited by specific Ca(2+) channel blockade
KEY POINTS: Bradykinin may play a role in the autodigestive disease acute pancreatitis, but little is known about its pancreatic actions. In this study, we have investigated bradykinin‐elicited Ca(2+) signal generation in normal mouse pancreatic lobules. We found complete separation of Ca(2+) signal...
Autores principales: | Gryshchenko, Oleksiy, Gerasimenko, Julia V., Gerasimenko, Oleg V., Petersen, Ole H. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4713750/ https://www.ncbi.nlm.nih.gov/pubmed/26442817 http://dx.doi.org/10.1113/JP271468 |
Ejemplares similares
-
SARS-CoV-2 S Protein Subunit 1 Elicits Ca(2+) Influx – Dependent Ca(2+) Signals in Pancreatic Stellate Cells and Macrophages In Situ
por: Gerasimenko, Julia V, et al.
Publicado: (2022) -
Bile acids induce necrosis in pancreatic stellate cells dependent on calcium entry and sodium‐driven bile uptake
por: Ferdek, Pawel E., et al.
Publicado: (2016) -
The role of Ca(2+) in the pathophysiology of pancreatitis
por: Gerasimenko, Julia V, et al.
Publicado: (2014) -
Calcium Signaling in Pancreatic Immune Cells In situ
por: Gryshchenko, Oleksiy, et al.
Publicado: (2020) -
Calcium and adenosine triphosphate control of cellular pathology: asparaginase-induced pancreatitis elicited via protease-activated receptor 2
por: Peng, Shuang, et al.
Publicado: (2016)