Cargando…

“Less is more”: Non operative management of short term superior mesenteric artery syndrome

INTRODUCTION: Superior mesenteric artery (SMA) syndrome is a relatively rare aetiology of proximal intestinal obstruction. This is caused by narrowing of vascular angle of SMA and aorta compressing the third part of the duodenum (D3). Predisposing factors may include precipitous weight loss, correct...

Descripción completa

Detalles Bibliográficos
Autores principales: Naseem, Zainab, Premaratne, Gamini, Hendahewa, Rasika
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Elsevier 2015
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4720713/
https://www.ncbi.nlm.nih.gov/pubmed/26904194
http://dx.doi.org/10.1016/j.amsu.2015.10.011
_version_ 1782411110065897472
author Naseem, Zainab
Premaratne, Gamini
Hendahewa, Rasika
author_facet Naseem, Zainab
Premaratne, Gamini
Hendahewa, Rasika
author_sort Naseem, Zainab
collection PubMed
description INTRODUCTION: Superior mesenteric artery (SMA) syndrome is a relatively rare aetiology of proximal intestinal obstruction. This is caused by narrowing of vascular angle of SMA and aorta compressing the third part of the duodenum (D3). Predisposing factors may include precipitous weight loss, corrective spinal surgery or repair of an aortic aneurysm. PRESENTATION OF CASE: A 53 year old male presented to our department with worsening post-prandial vomiting and epigastric pain for last three months. One examination, epigastric region was distended with succussion splash on abdominal auscultation. History did not include any predisposing factor. CT scan showed narrow angle of 12.77° between SMA and aorta owing to the compression of D3. Since onset of vomiting and resultant poor oral intake, he had lost 11 kg of his usual body weight. After gastric decompression, nasojejunal enteral feeding was started. Diet was progressed to oral feedings gradually and following return to his baseline weight, he continued to be free of symptoms in follow-up visits. DISCUSSION: Although there are recognised predisposing factors but sometimes aetiology remains idiopathic. SMA syndrome should initially be managed non-operatively with gastric decompression, correction of water and electrolyte imbalance, and hemodynamic instability. Regaining weight helps increasing vascular space between SMA and D3 thus relieving obstruction. Persistence of symptoms beyond 3–4 weeks warrants surgical intervention. CONCLUSION: Non operative management with nutritional supplementation remains the first line of therapy.
format Online
Article
Text
id pubmed-4720713
institution National Center for Biotechnology Information
language English
publishDate 2015
publisher Elsevier
record_format MEDLINE/PubMed
spelling pubmed-47207132016-02-22 “Less is more”: Non operative management of short term superior mesenteric artery syndrome Naseem, Zainab Premaratne, Gamini Hendahewa, Rasika Ann Med Surg (Lond) Case Report INTRODUCTION: Superior mesenteric artery (SMA) syndrome is a relatively rare aetiology of proximal intestinal obstruction. This is caused by narrowing of vascular angle of SMA and aorta compressing the third part of the duodenum (D3). Predisposing factors may include precipitous weight loss, corrective spinal surgery or repair of an aortic aneurysm. PRESENTATION OF CASE: A 53 year old male presented to our department with worsening post-prandial vomiting and epigastric pain for last three months. One examination, epigastric region was distended with succussion splash on abdominal auscultation. History did not include any predisposing factor. CT scan showed narrow angle of 12.77° between SMA and aorta owing to the compression of D3. Since onset of vomiting and resultant poor oral intake, he had lost 11 kg of his usual body weight. After gastric decompression, nasojejunal enteral feeding was started. Diet was progressed to oral feedings gradually and following return to his baseline weight, he continued to be free of symptoms in follow-up visits. DISCUSSION: Although there are recognised predisposing factors but sometimes aetiology remains idiopathic. SMA syndrome should initially be managed non-operatively with gastric decompression, correction of water and electrolyte imbalance, and hemodynamic instability. Regaining weight helps increasing vascular space between SMA and D3 thus relieving obstruction. Persistence of symptoms beyond 3–4 weeks warrants surgical intervention. CONCLUSION: Non operative management with nutritional supplementation remains the first line of therapy. Elsevier 2015-10-23 /pmc/articles/PMC4720713/ /pubmed/26904194 http://dx.doi.org/10.1016/j.amsu.2015.10.011 Text en © 2015 The Authors http://creativecommons.org/licenses/by-nc-nd/4.0/ This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).
spellingShingle Case Report
Naseem, Zainab
Premaratne, Gamini
Hendahewa, Rasika
“Less is more”: Non operative management of short term superior mesenteric artery syndrome
title “Less is more”: Non operative management of short term superior mesenteric artery syndrome
title_full “Less is more”: Non operative management of short term superior mesenteric artery syndrome
title_fullStr “Less is more”: Non operative management of short term superior mesenteric artery syndrome
title_full_unstemmed “Less is more”: Non operative management of short term superior mesenteric artery syndrome
title_short “Less is more”: Non operative management of short term superior mesenteric artery syndrome
title_sort “less is more”: non operative management of short term superior mesenteric artery syndrome
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4720713/
https://www.ncbi.nlm.nih.gov/pubmed/26904194
http://dx.doi.org/10.1016/j.amsu.2015.10.011
work_keys_str_mv AT naseemzainab lessismorenonoperativemanagementofshorttermsuperiormesentericarterysyndrome
AT premaratnegamini lessismorenonoperativemanagementofshorttermsuperiormesentericarterysyndrome
AT hendahewarasika lessismorenonoperativemanagementofshorttermsuperiormesentericarterysyndrome