Freezing of gait associated with a corpus callosum lesion

Freezing of gait (FoG) is a debilitating feature of Parkinson’s disease and other parkinsonian disorders. This case demonstrates a variant of freezing of gait in a non-parkinsonian patient with a lesion of the anterior corpus callosum. The freezing improved with increased upper extremity sensory inp...

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Autores principales: Dale, Marian L., Mancini, Martina, Curtze, Carolin, Horak, Fay B., Fling, Brett W.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2016
Materias:
Case Report
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4731964/
https://www.ncbi.nlm.nih.gov/pubmed/26835154
http://dx.doi.org/10.1186/s40734-016-0030-2
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author Dale, Marian L.
Mancini, Martina
Curtze, Carolin
Horak, Fay B.
Fling, Brett W.
author_facet Dale, Marian L.
Mancini, Martina
Curtze, Carolin
Horak, Fay B.
Fling, Brett W.
author_sort Dale, Marian L.
collection PubMed
description Freezing of gait (FoG) is a debilitating feature of Parkinson’s disease and other parkinsonian disorders. This case demonstrates a variant of freezing of gait in a non-parkinsonian patient with a lesion of the anterior corpus callosum. The freezing improved with increased upper extremity sensory input, suggesting that compensatory circuits for use of somatosensory inputs from the arms to postural and locomotor centers were intact. ELECTRONIC SUPPLEMENTARY MATERIAL: The online version of this article (doi:10.1186/s40734-016-0030-2) contains supplementary material, which is available to authorized users.
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spelling pubmed-47319642016-01-30 Freezing of gait associated with a corpus callosum lesion Dale, Marian L. Mancini, Martina Curtze, Carolin Horak, Fay B. Fling, Brett W. J Clin Mov Disord Case Report Freezing of gait (FoG) is a debilitating feature of Parkinson’s disease and other parkinsonian disorders. This case demonstrates a variant of freezing of gait in a non-parkinsonian patient with a lesion of the anterior corpus callosum. The freezing improved with increased upper extremity sensory input, suggesting that compensatory circuits for use of somatosensory inputs from the arms to postural and locomotor centers were intact. ELECTRONIC SUPPLEMENTARY MATERIAL: The online version of this article (doi:10.1186/s40734-016-0030-2) contains supplementary material, which is available to authorized users. BioMed Central 2016-01-29 /pmc/articles/PMC4731964/ /pubmed/26835154 http://dx.doi.org/10.1186/s40734-016-0030-2 Text en © Dale et al. 2016 Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.
spellingShingle Case Report
Dale, Marian L.
Mancini, Martina
Curtze, Carolin
Horak, Fay B.
Fling, Brett W.
Freezing of gait associated with a corpus callosum lesion
title Freezing of gait associated with a corpus callosum lesion
title_full Freezing of gait associated with a corpus callosum lesion
title_fullStr Freezing of gait associated with a corpus callosum lesion
title_full_unstemmed Freezing of gait associated with a corpus callosum lesion
title_short Freezing of gait associated with a corpus callosum lesion
title_sort freezing of gait associated with a corpus callosum lesion
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4731964/
https://www.ncbi.nlm.nih.gov/pubmed/26835154
http://dx.doi.org/10.1186/s40734-016-0030-2
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