Propofol Suppressed Hypoxia/Reoxygenation-Induced Apoptosis in HBVSMC by Regulation of the Expression of Bcl-2, Bax, Caspase3, Kir6.1, and p-JNK

Recent studies have found that propofol may protect brain from cerebral ischemic-reperfusion injury. However, the underlying mechanism remains unclear. The effects of propofol were evaluated in HBVSMC after hypoxia/reoxygenation (H/R). Cell viability and levels of SOD, LDH, and MDA were measured. Ap...

Descripción completa

Detalles Bibliográficos
Autores principales: Zhang, Jianhai, Xia, Yunfei, Xu, Zifeng, Deng, Xiaoming
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi Publishing Corporation 2016
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4736333/
https://www.ncbi.nlm.nih.gov/pubmed/27057270
http://dx.doi.org/10.1155/2016/1518738

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4736333/
https://www.ncbi.nlm.nih.gov/pubmed/27057270
http://dx.doi.org/10.1155/2016/1518738

Ejemplares similares