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Neuroimaging evidence of deficient axon myelination in Wolfram syndrome

Wolfram syndrome is a rare autosomal recessive genetic disease characterized by insulin dependent diabetes and vision, hearing and brain abnormalities which generally emerge in childhood. Mutations in the WFS1 gene predispose cells to endoplasmic reticulum stress-mediated apoptosis and may induce my...

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Autores principales: Lugar, Heather M., Koller, Jonathan M., Rutlin, Jerrel, Marshall, Bess A., Kanekura, Kohsuke, Urano, Fumihiko, Bischoff, Allison N., Shimony, Joshua S., Hershey, Tamara
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2016
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4758056/
https://www.ncbi.nlm.nih.gov/pubmed/26888576
http://dx.doi.org/10.1038/srep21167
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author Lugar, Heather M.
Koller, Jonathan M.
Rutlin, Jerrel
Marshall, Bess A.
Kanekura, Kohsuke
Urano, Fumihiko
Bischoff, Allison N.
Shimony, Joshua S.
Hershey, Tamara
author_facet Lugar, Heather M.
Koller, Jonathan M.
Rutlin, Jerrel
Marshall, Bess A.
Kanekura, Kohsuke
Urano, Fumihiko
Bischoff, Allison N.
Shimony, Joshua S.
Hershey, Tamara
author_sort Lugar, Heather M.
collection PubMed
description Wolfram syndrome is a rare autosomal recessive genetic disease characterized by insulin dependent diabetes and vision, hearing and brain abnormalities which generally emerge in childhood. Mutations in the WFS1 gene predispose cells to endoplasmic reticulum stress-mediated apoptosis and may induce myelin degradation in neuronal cell models. However, in vivo evidence of this phenomenon in humans is lacking. White matter microstructure and regional volumes were measured using magnetic resonance imaging in children and young adults with Wolfram syndrome (n = 21) and healthy and diabetic controls (n = 50). Wolfram patients had lower fractional anisotropy and higher radial diffusivity in major white matter tracts and lower volume in the basilar (ventral) pons, cerebellar white matter and visual cortex. Correlations were found between key brain findings and overall neurological symptoms. This pattern of findings suggests that reduction in myelin is a primary neuropathological feature of Wolfram syndrome. Endoplasmic reticulum stress-related dysfunction in Wolfram syndrome may interact with the development of myelin or promote degeneration of myelin during the progression of the disease. These measures may provide objective indices of Wolfram syndrome pathophysiology that will be useful in unraveling the underlying mechanisms and in testing the impact of treatments on the brain.
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spelling pubmed-47580562016-02-26 Neuroimaging evidence of deficient axon myelination in Wolfram syndrome Lugar, Heather M. Koller, Jonathan M. Rutlin, Jerrel Marshall, Bess A. Kanekura, Kohsuke Urano, Fumihiko Bischoff, Allison N. Shimony, Joshua S. Hershey, Tamara Sci Rep Article Wolfram syndrome is a rare autosomal recessive genetic disease characterized by insulin dependent diabetes and vision, hearing and brain abnormalities which generally emerge in childhood. Mutations in the WFS1 gene predispose cells to endoplasmic reticulum stress-mediated apoptosis and may induce myelin degradation in neuronal cell models. However, in vivo evidence of this phenomenon in humans is lacking. White matter microstructure and regional volumes were measured using magnetic resonance imaging in children and young adults with Wolfram syndrome (n = 21) and healthy and diabetic controls (n = 50). Wolfram patients had lower fractional anisotropy and higher radial diffusivity in major white matter tracts and lower volume in the basilar (ventral) pons, cerebellar white matter and visual cortex. Correlations were found between key brain findings and overall neurological symptoms. This pattern of findings suggests that reduction in myelin is a primary neuropathological feature of Wolfram syndrome. Endoplasmic reticulum stress-related dysfunction in Wolfram syndrome may interact with the development of myelin or promote degeneration of myelin during the progression of the disease. These measures may provide objective indices of Wolfram syndrome pathophysiology that will be useful in unraveling the underlying mechanisms and in testing the impact of treatments on the brain. Nature Publishing Group 2016-02-18 /pmc/articles/PMC4758056/ /pubmed/26888576 http://dx.doi.org/10.1038/srep21167 Text en Copyright © 2016, Macmillan Publishers Limited http://creativecommons.org/licenses/by/4.0/ This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/
spellingShingle Article
Lugar, Heather M.
Koller, Jonathan M.
Rutlin, Jerrel
Marshall, Bess A.
Kanekura, Kohsuke
Urano, Fumihiko
Bischoff, Allison N.
Shimony, Joshua S.
Hershey, Tamara
Neuroimaging evidence of deficient axon myelination in Wolfram syndrome
title Neuroimaging evidence of deficient axon myelination in Wolfram syndrome
title_full Neuroimaging evidence of deficient axon myelination in Wolfram syndrome
title_fullStr Neuroimaging evidence of deficient axon myelination in Wolfram syndrome
title_full_unstemmed Neuroimaging evidence of deficient axon myelination in Wolfram syndrome
title_short Neuroimaging evidence of deficient axon myelination in Wolfram syndrome
title_sort neuroimaging evidence of deficient axon myelination in wolfram syndrome
topic Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4758056/
https://www.ncbi.nlm.nih.gov/pubmed/26888576
http://dx.doi.org/10.1038/srep21167
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