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The etiology of primary femoroacetabular impingement: genetics or acquired deformity?

The etiology of primary femoroacetabular impingement (FAI) remains controversial. Both genetic and acquired causes have been postulated and studied. While recent studies suggest that genetic factors may have a role in the development of FAI, there is no conclusive evidence that FAI is transmitted ge...

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Autores principales: Packer, Jonathan D., Safran, Marc R.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Oxford University Press 2015
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4765309/
https://www.ncbi.nlm.nih.gov/pubmed/27011846
http://dx.doi.org/10.1093/jhps/hnv046
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author Packer, Jonathan D.
Safran, Marc R.
author_facet Packer, Jonathan D.
Safran, Marc R.
author_sort Packer, Jonathan D.
collection PubMed
description The etiology of primary femoroacetabular impingement (FAI) remains controversial. Both genetic and acquired causes have been postulated and studied. While recent studies suggest that genetic factors may have a role in the development of FAI, there is no conclusive evidence that FAI is transmitted genetically. Currently, the most popular theory for the development of cam-type deformities is that a repetitive injury to the proximal femoral physis occurs during a critical period of development. There is a correlation between a high volume of impact activities during adolescence and the development of cam-type deformities. Multiple studies have found a high prevalence of FAI in elite football, ice hockey, basketball and soccer players. In this article, we review the current literature relating to the etiology of primary FAI.
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spelling pubmed-47653092016-03-23 The etiology of primary femoroacetabular impingement: genetics or acquired deformity? Packer, Jonathan D. Safran, Marc R. J Hip Preserv Surg Review Articles The etiology of primary femoroacetabular impingement (FAI) remains controversial. Both genetic and acquired causes have been postulated and studied. While recent studies suggest that genetic factors may have a role in the development of FAI, there is no conclusive evidence that FAI is transmitted genetically. Currently, the most popular theory for the development of cam-type deformities is that a repetitive injury to the proximal femoral physis occurs during a critical period of development. There is a correlation between a high volume of impact activities during adolescence and the development of cam-type deformities. Multiple studies have found a high prevalence of FAI in elite football, ice hockey, basketball and soccer players. In this article, we review the current literature relating to the etiology of primary FAI. Oxford University Press 2015-06-18 /pmc/articles/PMC4765309/ /pubmed/27011846 http://dx.doi.org/10.1093/jhps/hnv046 Text en © The Author 2015. Published by Oxford University Press http://creativecommons.org/licenses/by/4.0/ This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Review Articles
Packer, Jonathan D.
Safran, Marc R.
The etiology of primary femoroacetabular impingement: genetics or acquired deformity?
title The etiology of primary femoroacetabular impingement: genetics or acquired deformity?
title_full The etiology of primary femoroacetabular impingement: genetics or acquired deformity?
title_fullStr The etiology of primary femoroacetabular impingement: genetics or acquired deformity?
title_full_unstemmed The etiology of primary femoroacetabular impingement: genetics or acquired deformity?
title_short The etiology of primary femoroacetabular impingement: genetics or acquired deformity?
title_sort etiology of primary femoroacetabular impingement: genetics or acquired deformity?
topic Review Articles
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4765309/
https://www.ncbi.nlm.nih.gov/pubmed/27011846
http://dx.doi.org/10.1093/jhps/hnv046
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