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Genome-wide analysis of the specificity and mechanisms of replication infidelity driven by imbalanced dNTP pools
The absolute and relative concentrations of the four dNTPs are key determinants of DNA replication fidelity, yet the consequences of altered dNTP pools on replication fidelity have not previously been investigated on a genome-wide scale. Here, we use deep sequencing to determine the types, rates and...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Oxford University Press
2016
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4770217/ https://www.ncbi.nlm.nih.gov/pubmed/26609135 http://dx.doi.org/10.1093/nar/gkv1298 |
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author | Watt, Danielle L. Buckland, Robert J. Lujan, Scott A. Kunkel, Thomas A. Chabes, Andrei |
author_facet | Watt, Danielle L. Buckland, Robert J. Lujan, Scott A. Kunkel, Thomas A. Chabes, Andrei |
author_sort | Watt, Danielle L. |
collection | PubMed |
description | The absolute and relative concentrations of the four dNTPs are key determinants of DNA replication fidelity, yet the consequences of altered dNTP pools on replication fidelity have not previously been investigated on a genome-wide scale. Here, we use deep sequencing to determine the types, rates and locations of uncorrected replication errors that accumulate in the nuclear genome of a mismatch repair-deficient diploid yeast strain with elevated dCTP and dTTP concentrations. These imbalanced dNTP pools promote replication errors in specific DNA sequence motifs suggesting increased misinsertion and increased mismatch extension at the expense of proofreading. Interestingly, substitution rates are similar for leading and lagging strand replication, but are higher in regions replicated late in S phase. Remarkably, the rate of single base deletions is preferentially increased in coding sequences and in short rather than long mononucleotides runs. Based on DNA sequence motifs, we propose two distinct mechanisms for generating single base deletions in vivo. Collectively, the results indicate that elevated dCTP and dTTP pools increase mismatch formation and decrease error correction across the nuclear genome, and most strongly increases mutation rates in coding and late replicating sequences. |
format | Online Article Text |
id | pubmed-4770217 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2016 |
publisher | Oxford University Press |
record_format | MEDLINE/PubMed |
spelling | pubmed-47702172016-02-29 Genome-wide analysis of the specificity and mechanisms of replication infidelity driven by imbalanced dNTP pools Watt, Danielle L. Buckland, Robert J. Lujan, Scott A. Kunkel, Thomas A. Chabes, Andrei Nucleic Acids Res Genome Integrity, Repair and Replication The absolute and relative concentrations of the four dNTPs are key determinants of DNA replication fidelity, yet the consequences of altered dNTP pools on replication fidelity have not previously been investigated on a genome-wide scale. Here, we use deep sequencing to determine the types, rates and locations of uncorrected replication errors that accumulate in the nuclear genome of a mismatch repair-deficient diploid yeast strain with elevated dCTP and dTTP concentrations. These imbalanced dNTP pools promote replication errors in specific DNA sequence motifs suggesting increased misinsertion and increased mismatch extension at the expense of proofreading. Interestingly, substitution rates are similar for leading and lagging strand replication, but are higher in regions replicated late in S phase. Remarkably, the rate of single base deletions is preferentially increased in coding sequences and in short rather than long mononucleotides runs. Based on DNA sequence motifs, we propose two distinct mechanisms for generating single base deletions in vivo. Collectively, the results indicate that elevated dCTP and dTTP pools increase mismatch formation and decrease error correction across the nuclear genome, and most strongly increases mutation rates in coding and late replicating sequences. Oxford University Press 2016-02-29 2015-11-24 /pmc/articles/PMC4770217/ /pubmed/26609135 http://dx.doi.org/10.1093/nar/gkv1298 Text en © The Author(s) 2015. Published by Oxford University Press on behalf of Nucleic Acids Research. http://creativecommons.org/licenses/by-nc/4.0/ This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com |
spellingShingle | Genome Integrity, Repair and Replication Watt, Danielle L. Buckland, Robert J. Lujan, Scott A. Kunkel, Thomas A. Chabes, Andrei Genome-wide analysis of the specificity and mechanisms of replication infidelity driven by imbalanced dNTP pools |
title | Genome-wide analysis of the specificity and mechanisms of replication infidelity driven by imbalanced dNTP pools |
title_full | Genome-wide analysis of the specificity and mechanisms of replication infidelity driven by imbalanced dNTP pools |
title_fullStr | Genome-wide analysis of the specificity and mechanisms of replication infidelity driven by imbalanced dNTP pools |
title_full_unstemmed | Genome-wide analysis of the specificity and mechanisms of replication infidelity driven by imbalanced dNTP pools |
title_short | Genome-wide analysis of the specificity and mechanisms of replication infidelity driven by imbalanced dNTP pools |
title_sort | genome-wide analysis of the specificity and mechanisms of replication infidelity driven by imbalanced dntp pools |
topic | Genome Integrity, Repair and Replication |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4770217/ https://www.ncbi.nlm.nih.gov/pubmed/26609135 http://dx.doi.org/10.1093/nar/gkv1298 |
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