High dietary advanced glycation end products are associated with poorer spatial learning and accelerated Aβ deposition in an Alzheimer mouse model

There is growing evidence of the involvement of advanced glycation end products (AGEs) in the pathogenesis of neurodegenerative processes including Alzheimer's disease (AD) and their function as a seed for the aggregation of Aβ, a hallmark feature of AD. AGEs are formed endogenously and exogeno...

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Detalles Bibliográficos
Autores principales: Lubitz, Irit, Ricny, Jan, Atrakchi‐Baranes, Dana, Shemesh, Chen, Kravitz, Efrat, Liraz‐Zaltsman, Sigal, Maksin‐Matveev, Anna, Cooper, Itzik, Leibowitz, Avshalom, Uribarri, Jaime, Schmeidler, James, Cai, Weijing, Kristofikova, Zdena, Ripova, Daniela, LeRoith, Derek, Schnaider‐Beeri, Michal
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2016
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4783343/
https://www.ncbi.nlm.nih.gov/pubmed/26781037
http://dx.doi.org/10.1111/acel.12436

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