Collapsing glomerulopathy in a case of anti-neutrophil cytoplasmic antibody associated vasculitis

Collapsing glomerulopathy (CG) is a pathological entity characterized by collapse and wrinkling of glomerular tuft, podocyte dedifferentiation and hyperplasia. CG may be idiopathic or secondary to other diseases. CG has been described with IgA nephropathy, membranous glomerulopathy, diabetic nephrop...

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Autores principales: Singh, N., Rathi, M., Nada, R., Sharma, A., Goyal, A., Ramachandran, R., Kumar, V., Kohli, H. S., Gupta, K. L.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Medknow Publications & Media Pvt Ltd 2016
Materias:
Case Report
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4795431/
https://www.ncbi.nlm.nih.gov/pubmed/27051140
http://dx.doi.org/10.4103/0971-4065.161022
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author Singh, N.
Rathi, M.
Nada, R.
Sharma, A.
Goyal, A.
Ramachandran, R.
Kumar, V.
Kohli, H. S.
Gupta, K. L.
author_facet Singh, N.
Rathi, M.
Nada, R.
Sharma, A.
Goyal, A.
Ramachandran, R.
Kumar, V.
Kohli, H. S.
Gupta, K. L.
author_sort Singh, N.
collection PubMed
description Collapsing glomerulopathy (CG) is a pathological entity characterized by collapse and wrinkling of glomerular tuft, podocyte dedifferentiation and hyperplasia. CG may be idiopathic or secondary to other diseases. CG has been described with IgA nephropathy, membranous glomerulopathy, diabetic nephropathy, and lupus nephritis. However, till date there is no report of CG in association with the anti-neutrophil cytoplasmic antibody (ANCA) associated vasculitis (AAV). Here, we present a case of CG that developed during follow-up in a case of AAV with biopsy proven pauci-immune glomerulonephritis.
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spelling pubmed-47954312016-04-05 Collapsing glomerulopathy in a case of anti-neutrophil cytoplasmic antibody associated vasculitis Singh, N. Rathi, M. Nada, R. Sharma, A. Goyal, A. Ramachandran, R. Kumar, V. Kohli, H. S. Gupta, K. L. Indian J Nephrol Case Report Collapsing glomerulopathy (CG) is a pathological entity characterized by collapse and wrinkling of glomerular tuft, podocyte dedifferentiation and hyperplasia. CG may be idiopathic or secondary to other diseases. CG has been described with IgA nephropathy, membranous glomerulopathy, diabetic nephropathy, and lupus nephritis. However, till date there is no report of CG in association with the anti-neutrophil cytoplasmic antibody (ANCA) associated vasculitis (AAV). Here, we present a case of CG that developed during follow-up in a case of AAV with biopsy proven pauci-immune glomerulonephritis. Medknow Publications & Media Pvt Ltd 2016 /pmc/articles/PMC4795431/ /pubmed/27051140 http://dx.doi.org/10.4103/0971-4065.161022 Text en Copyright: © 2016 Indian Journal of Nephrology http://creativecommons.org/licenses/by-nc-sa/3.0 This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 3.0 License, which allows others to remix, tweak, and build upon the work non-commercially, as long as the author is credited and the new creations are licensed under the identical terms.
spellingShingle Case Report
Singh, N.
Rathi, M.
Nada, R.
Sharma, A.
Goyal, A.
Ramachandran, R.
Kumar, V.
Kohli, H. S.
Gupta, K. L.
Collapsing glomerulopathy in a case of anti-neutrophil cytoplasmic antibody associated vasculitis
title Collapsing glomerulopathy in a case of anti-neutrophil cytoplasmic antibody associated vasculitis
title_full Collapsing glomerulopathy in a case of anti-neutrophil cytoplasmic antibody associated vasculitis
title_fullStr Collapsing glomerulopathy in a case of anti-neutrophil cytoplasmic antibody associated vasculitis
title_full_unstemmed Collapsing glomerulopathy in a case of anti-neutrophil cytoplasmic antibody associated vasculitis
title_short Collapsing glomerulopathy in a case of anti-neutrophil cytoplasmic antibody associated vasculitis
title_sort collapsing glomerulopathy in a case of anti-neutrophil cytoplasmic antibody associated vasculitis
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4795431/
https://www.ncbi.nlm.nih.gov/pubmed/27051140
http://dx.doi.org/10.4103/0971-4065.161022
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