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Excitatory transmission onto AgRP neurons is regulated by cJun NH(2)-terminal kinase 3 in response to metabolic stress

The cJun NH(2)-terminal kinase (JNK) signaling pathway is implicated in the response to metabolic stress. Indeed, it is established that the ubiquitously expressed JNK1 and JNK2 isoforms regulate energy expenditure and insulin resistance. However, the role of the neuron-specific isoform JNK3 is uncl...

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Detalles Bibliográficos
Autores principales: Vernia, Santiago, Morel, Caroline, Madara, Joseph C, Cavanagh-Kyros, Julie, Barrett, Tamera, Chase, Kathryn, Kennedy, Norman J, Jung, Dae Young, Kim, Jason K, Aronin, Neil, Flavell, Richard A, Lowell, Bradford B, Davis, Roger J
Formato: Online Artículo Texto
Lenguaje:English
Publicado: eLife Sciences Publications, Ltd 2016
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4798947/
https://www.ncbi.nlm.nih.gov/pubmed/26910012
http://dx.doi.org/10.7554/eLife.10031

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