Cargando…
Response to “ROS, P53 and ischemic acute kidney injury in diabetic models”
Autor principal: | Dong, Zheng |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
2015
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4811037/ https://www.ncbi.nlm.nih.gov/pubmed/26126101 http://dx.doi.org/10.1038/ki.2015.131 |
Ejemplares similares
-
Hyperglycemia, p53 and mitochondrial pathway of apoptosis are involved in the susceptibility of diabetic models to ischemic acute kidney injury
por: Peng, Jianping, et al.
Publicado: (2014) -
Hexarelin alleviates apoptosis on ischemic acute kidney injury via MDM2/p53 pathway
por: Guan, Chen, et al.
Publicado: (2023) -
Mitochondria ROS and mitophagy in acute kidney injury
por: Su, Lianjiu, et al.
Publicado: (2022) -
p53, ROS and senescence in the control of aging
por: Vigneron, Arnaud, et al.
Publicado: (2010) -
Pyrocatechol Alleviates Cisplatin-Induced Acute Kidney Injury by Inhibiting ROS Production
por: Xie, Xuexia, et al.
Publicado: (2022)