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The Role of miR-34a in Tritiated Water Toxicity in Human Umbilical Vein Endothelial Cells
In this work, we investigated the toxic effects of tritiated water (HTO) on the cardiovascular system. We examined the role of microRNA-34a (miR-34a) in DNA damage and repair in human umbilical vein endothelial cells (HUVECs) exposed to HTO. Cell proliferation capacity was evaluated by cell counting...
Autores principales: | , , , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
SAGE Publications
2016
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4822198/ https://www.ncbi.nlm.nih.gov/pubmed/27099602 http://dx.doi.org/10.1177/1559325816638585 |
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author | Cui, Feng Mei Liu, Liang Zheng, Lu Lin Bao, Guang Liang Tu, Yu Sun, Liang Zhu, Wei Cao, Jian Ping Zhou, Ping Kun Chen, Qiu He, Yong Ming |
author_facet | Cui, Feng Mei Liu, Liang Zheng, Lu Lin Bao, Guang Liang Tu, Yu Sun, Liang Zhu, Wei Cao, Jian Ping Zhou, Ping Kun Chen, Qiu He, Yong Ming |
author_sort | Cui, Feng Mei |
collection | PubMed |
description | In this work, we investigated the toxic effects of tritiated water (HTO) on the cardiovascular system. We examined the role of microRNA-34a (miR-34a) in DNA damage and repair in human umbilical vein endothelial cells (HUVECs) exposed to HTO. Cell proliferation capacity was evaluated by cell counting, and miR-34a expression was detected using quantitative PCR (QT-PCR). The Comet assay and γ-H2AX immunostaining were used to measure DNA double-strand breaks (DSBs). Reverse transcription polymerase chain reaction was used to measure the expression level of c-myc messenger RNA (mRNA). The cells exposed to HTO showed significantly lower proliferation than the control cells over 3 days. The DNA damage in the HTO group was more severe than that in the control group, at each time point examined. The expression of miR-34a mimics caused increased DNA DSBs whereas that of the miR-34a inhibitor caused decreased DNA DSBs. The proliferation viability was the opposite for the miR-34a mimics and inhibitor groups. The expression levels of c-myc mRNA in cells transfected with miR-34a mimics were lower than that in cells transfected with the miR-34a-5p inhibitor, at 0.5 hours and 2 hours after transfection. In summary, miR-34a mediates HTO toxicity in HUVECs by downregulating the expression of c-myc. |
format | Online Article Text |
id | pubmed-4822198 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2016 |
publisher | SAGE Publications |
record_format | MEDLINE/PubMed |
spelling | pubmed-48221982016-04-20 The Role of miR-34a in Tritiated Water Toxicity in Human Umbilical Vein Endothelial Cells Cui, Feng Mei Liu, Liang Zheng, Lu Lin Bao, Guang Liang Tu, Yu Sun, Liang Zhu, Wei Cao, Jian Ping Zhou, Ping Kun Chen, Qiu He, Yong Ming Dose Response Original Articles In this work, we investigated the toxic effects of tritiated water (HTO) on the cardiovascular system. We examined the role of microRNA-34a (miR-34a) in DNA damage and repair in human umbilical vein endothelial cells (HUVECs) exposed to HTO. Cell proliferation capacity was evaluated by cell counting, and miR-34a expression was detected using quantitative PCR (QT-PCR). The Comet assay and γ-H2AX immunostaining were used to measure DNA double-strand breaks (DSBs). Reverse transcription polymerase chain reaction was used to measure the expression level of c-myc messenger RNA (mRNA). The cells exposed to HTO showed significantly lower proliferation than the control cells over 3 days. The DNA damage in the HTO group was more severe than that in the control group, at each time point examined. The expression of miR-34a mimics caused increased DNA DSBs whereas that of the miR-34a inhibitor caused decreased DNA DSBs. The proliferation viability was the opposite for the miR-34a mimics and inhibitor groups. The expression levels of c-myc mRNA in cells transfected with miR-34a mimics were lower than that in cells transfected with the miR-34a-5p inhibitor, at 0.5 hours and 2 hours after transfection. In summary, miR-34a mediates HTO toxicity in HUVECs by downregulating the expression of c-myc. SAGE Publications 2016-03-30 /pmc/articles/PMC4822198/ /pubmed/27099602 http://dx.doi.org/10.1177/1559325816638585 Text en © The Author(s) 2016 http://creativecommons.org/licenses/by-nc/3.0/ This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 3.0 License (http://www.creativecommons.org/licenses/by-nc/3.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage). |
spellingShingle | Original Articles Cui, Feng Mei Liu, Liang Zheng, Lu Lin Bao, Guang Liang Tu, Yu Sun, Liang Zhu, Wei Cao, Jian Ping Zhou, Ping Kun Chen, Qiu He, Yong Ming The Role of miR-34a in Tritiated Water Toxicity in Human Umbilical Vein Endothelial Cells |
title | The Role of miR-34a in Tritiated Water Toxicity in Human Umbilical Vein Endothelial Cells |
title_full | The Role of miR-34a in Tritiated Water Toxicity in Human Umbilical Vein Endothelial Cells |
title_fullStr | The Role of miR-34a in Tritiated Water Toxicity in Human Umbilical Vein Endothelial Cells |
title_full_unstemmed | The Role of miR-34a in Tritiated Water Toxicity in Human Umbilical Vein Endothelial Cells |
title_short | The Role of miR-34a in Tritiated Water Toxicity in Human Umbilical Vein Endothelial Cells |
title_sort | role of mir-34a in tritiated water toxicity in human umbilical vein endothelial cells |
topic | Original Articles |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4822198/ https://www.ncbi.nlm.nih.gov/pubmed/27099602 http://dx.doi.org/10.1177/1559325816638585 |
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