Can targeting glutamate receptors with long-term heat acclimation improve outcomes following hypoxic injury?

Long-term heat acclimation appears to improve tolerance to hypoxic insults in various tissues, including brain, providing a promising avenue to improve functional outcomes following cerebrovascular events. Glutamate discharge is implicated in dysfunction following hypoxic stress and thus, targeting...

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Detalles Bibliográficos
Autores principales: Ely, Brett R, Brunt, Vienna E, Minson, Christopher T
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Taylor & Francis 2015
Materias:
Editorial Comment - Commissioned
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4843862/
https://www.ncbi.nlm.nih.gov/pubmed/27227003
http://dx.doi.org/10.4161/23328940.2014.992657
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author Ely, Brett R
Brunt, Vienna E
Minson, Christopher T
author_facet Ely, Brett R
Brunt, Vienna E
Minson, Christopher T
author_sort Ely, Brett R
collection PubMed
description Long-term heat acclimation appears to improve tolerance to hypoxic insults in various tissues, including brain, providing a promising avenue to improve functional outcomes following cerebrovascular events. Glutamate discharge is implicated in dysfunction following hypoxic stress and thus, targeting glutamate receptors with heat acclimation could improve cognitive outcomes following hypoxic injury.
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spelling pubmed-48438622016-05-25 Can targeting glutamate receptors with long-term heat acclimation improve outcomes following hypoxic injury? Ely, Brett R Brunt, Vienna E Minson, Christopher T Temperature (Austin) Editorial Comment - Commissioned Long-term heat acclimation appears to improve tolerance to hypoxic insults in various tissues, including brain, providing a promising avenue to improve functional outcomes following cerebrovascular events. Glutamate discharge is implicated in dysfunction following hypoxic stress and thus, targeting glutamate receptors with heat acclimation could improve cognitive outcomes following hypoxic injury. Taylor & Francis 2015-02-03 /pmc/articles/PMC4843862/ /pubmed/27227003 http://dx.doi.org/10.4161/23328940.2014.992657 Text en © 2015 The Author(s). Published with license by Taylor & Francis Group, LLC http://creativecommons.org/licenses/by-nc/3.0/ This is an Open Access article distributed under the terms of the Creative Commons Attribution-Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The moral rights of the named author(s) have been asserted.
spellingShingle Editorial Comment - Commissioned
Ely, Brett R
Brunt, Vienna E
Minson, Christopher T
Can targeting glutamate receptors with long-term heat acclimation improve outcomes following hypoxic injury?
title Can targeting glutamate receptors with long-term heat acclimation improve outcomes following hypoxic injury?
title_full Can targeting glutamate receptors with long-term heat acclimation improve outcomes following hypoxic injury?
title_fullStr Can targeting glutamate receptors with long-term heat acclimation improve outcomes following hypoxic injury?
title_full_unstemmed Can targeting glutamate receptors with long-term heat acclimation improve outcomes following hypoxic injury?
title_short Can targeting glutamate receptors with long-term heat acclimation improve outcomes following hypoxic injury?
title_sort can targeting glutamate receptors with long-term heat acclimation improve outcomes following hypoxic injury?
topic Editorial Comment - Commissioned
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4843862/
https://www.ncbi.nlm.nih.gov/pubmed/27227003
http://dx.doi.org/10.4161/23328940.2014.992657
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