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Interplay between hepatic mitochondria-associated membranes, lipid metabolism and caveolin-1 in mice

The mitochondria-associated membrane (MAM) is a specialized subdomain of the endoplasmic reticulum (ER) which acts as an intracellular signaling hub. MAM dysfunction has been related to liver disease. We report a high-throughput mass spectrometry-based proteomics characterization of MAMs from mouse...

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Detalles Bibliográficos
Autores principales: Sala-Vila, Aleix, Navarro-Lérida, Inmaculada, Sánchez-Alvarez, Miguel, Bosch, Marta, Calvo, Carlos, López, Juan Antonio, Calvo, Enrique, Ferguson, Charles, Giacomello, Marta, Serafini, Annalisa, Scorrano, Luca, Enriquez, José Antonio, Balsinde, Jesús, Parton, Robert G., Vázquez, Jesús, Pol, Albert, Del Pozo, Miguel A.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2016
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4894368/
https://www.ncbi.nlm.nih.gov/pubmed/27272971
http://dx.doi.org/10.1038/srep27351
Descripción
Sumario:The mitochondria-associated membrane (MAM) is a specialized subdomain of the endoplasmic reticulum (ER) which acts as an intracellular signaling hub. MAM dysfunction has been related to liver disease. We report a high-throughput mass spectrometry-based proteomics characterization of MAMs from mouse liver, which portrays them as an extremely complex compartment involved in different metabolic processes, including steroid metabolism. Interestingly, we identified caveolin-1 (CAV1) as an integral component of hepatic MAMs, which determine the relative cholesterol content of these ER subdomains. Finally, a detailed comparative proteomics analysis between MAMs from wild type and CAV1-deficient mice suggests that functional CAV1 contributes to the recruitment and regulation of intracellular steroid and lipoprotein metabolism-related processes accrued at MAMs. The potential impact of these novel aspects of CAV1 biology on global cell homeostasis and disease is discussed.