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Neuropathological assessment and validation of mouse models for Alzheimer's disease: applying NIA-AA guidelines

Dozens of transgenic mouse models, generally based on mutations associated with familial Alzheimer's disease (AD), have been developed, in part, for preclinical testing of candidate AD therapies. However, none of these models has successfully predicted the clinical efficacy of drugs for treatin...

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Autores principales: Keene, C. Dirk, Darvas, Martin, Kraemer, Brian, Liggitt, Denny, Sigurdson, Christina, Ladiges, Warren
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Co-Action Publishing 2016
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4912600/
https://www.ncbi.nlm.nih.gov/pubmed/27317189
http://dx.doi.org/10.3402/pba.v6.32397
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author Keene, C. Dirk
Darvas, Martin
Kraemer, Brian
Liggitt, Denny
Sigurdson, Christina
Ladiges, Warren
author_facet Keene, C. Dirk
Darvas, Martin
Kraemer, Brian
Liggitt, Denny
Sigurdson, Christina
Ladiges, Warren
author_sort Keene, C. Dirk
collection PubMed
description Dozens of transgenic mouse models, generally based on mutations associated with familial Alzheimer's disease (AD), have been developed, in part, for preclinical testing of candidate AD therapies. However, none of these models has successfully predicted the clinical efficacy of drugs for treating AD patients. Therefore, development of more translationally relevant AD mouse models remains a critical unmet need in the field. A concept not previously implemented in AD preclinical drug testing is the use of mouse lines that have been validated for neuropathological features of human AD. Current thinking suggests that amyloid plaque and neurofibrillary tangle deposition is an essential component for accurate modeling of AD. Therefore, the AD translational paradigm would require pathologic Aβ and tau deposition, a disease-relevant distribution of plaques and tangles, and a pattern of disease progression of Aβ and tau isoforms similar to the neuropathological features found in the brains of AD patients. Additional parameters useful to evaluate parallels between AD and animal models would include 1) cerebrospinal fluid (CSF) AD biomarker changes with reduced Aβ and increased phospho-tau/tau; 2) structural and functional neuroimaging patterns including MRI hippocampal atrophy, fluorodeoxyglucose (FDG), and amyloid/tau PET alterations in activity and/or patterns of pathologic peptide deposition and distribution; and 3) cognitive impairment with emphasis on spatial learning and memory to distinguish presymptomatic and symptomatic mice at specific ages. A validated AD mouse model for drug testing would likely show tau-related neurofibrillary degeneration following Aβ deposition and demonstrate changes in pathology, CSF analysis, and neuroimaging that mirror human AD. Development of the ideal model would revolutionize the ability to establish the translational value of AD mouse models and serve as a platform for discussions about national phenotyping guidelines and standards for models of AD and other neurodegenerative disorders.
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spelling pubmed-49126002016-07-06 Neuropathological assessment and validation of mouse models for Alzheimer's disease: applying NIA-AA guidelines Keene, C. Dirk Darvas, Martin Kraemer, Brian Liggitt, Denny Sigurdson, Christina Ladiges, Warren Pathobiol Aging Age Relat Dis Editorial Dozens of transgenic mouse models, generally based on mutations associated with familial Alzheimer's disease (AD), have been developed, in part, for preclinical testing of candidate AD therapies. However, none of these models has successfully predicted the clinical efficacy of drugs for treating AD patients. Therefore, development of more translationally relevant AD mouse models remains a critical unmet need in the field. A concept not previously implemented in AD preclinical drug testing is the use of mouse lines that have been validated for neuropathological features of human AD. Current thinking suggests that amyloid plaque and neurofibrillary tangle deposition is an essential component for accurate modeling of AD. Therefore, the AD translational paradigm would require pathologic Aβ and tau deposition, a disease-relevant distribution of plaques and tangles, and a pattern of disease progression of Aβ and tau isoforms similar to the neuropathological features found in the brains of AD patients. Additional parameters useful to evaluate parallels between AD and animal models would include 1) cerebrospinal fluid (CSF) AD biomarker changes with reduced Aβ and increased phospho-tau/tau; 2) structural and functional neuroimaging patterns including MRI hippocampal atrophy, fluorodeoxyglucose (FDG), and amyloid/tau PET alterations in activity and/or patterns of pathologic peptide deposition and distribution; and 3) cognitive impairment with emphasis on spatial learning and memory to distinguish presymptomatic and symptomatic mice at specific ages. A validated AD mouse model for drug testing would likely show tau-related neurofibrillary degeneration following Aβ deposition and demonstrate changes in pathology, CSF analysis, and neuroimaging that mirror human AD. Development of the ideal model would revolutionize the ability to establish the translational value of AD mouse models and serve as a platform for discussions about national phenotyping guidelines and standards for models of AD and other neurodegenerative disorders. Co-Action Publishing 2016-06-16 /pmc/articles/PMC4912600/ /pubmed/27317189 http://dx.doi.org/10.3402/pba.v6.32397 Text en © 2016 C. Dirk Keene et al. http://creativecommons.org/licenses/by-nc/4.0/ This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 International License, permitting all non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Editorial
Keene, C. Dirk
Darvas, Martin
Kraemer, Brian
Liggitt, Denny
Sigurdson, Christina
Ladiges, Warren
Neuropathological assessment and validation of mouse models for Alzheimer's disease: applying NIA-AA guidelines
title Neuropathological assessment and validation of mouse models for Alzheimer's disease: applying NIA-AA guidelines
title_full Neuropathological assessment and validation of mouse models for Alzheimer's disease: applying NIA-AA guidelines
title_fullStr Neuropathological assessment and validation of mouse models for Alzheimer's disease: applying NIA-AA guidelines
title_full_unstemmed Neuropathological assessment and validation of mouse models for Alzheimer's disease: applying NIA-AA guidelines
title_short Neuropathological assessment and validation of mouse models for Alzheimer's disease: applying NIA-AA guidelines
title_sort neuropathological assessment and validation of mouse models for alzheimer's disease: applying nia-aa guidelines
topic Editorial
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4912600/
https://www.ncbi.nlm.nih.gov/pubmed/27317189
http://dx.doi.org/10.3402/pba.v6.32397
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