Cargando…
Defects in MAP1S-mediated autophagy turnover of fibronectin cause renal fibrosis
Excessive deposition of extracellular matrix proteins in renal tissues causes renal fibrosis and renal function failure. Mammalian cells primarily use the autophagy-lysosome system to degrade misfolded/aggregated proteins and dysfunctional organelles. MAP1S is an autophagy activator and promotes the...
Autores principales: | Xu, Guibin, Yue, Fei, Huang, Hai, He, Yongzhong, Li, Xun, Zhao, Haibo, Su, Zhengming, Jiang, Xianhan, Li, Wenjiao, Zou, Jing, Chen, Qi, Liu, Leyuan |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Impact Journals LLC
2016
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4931848/ https://www.ncbi.nlm.nih.gov/pubmed/27236336 http://dx.doi.org/10.18632/aging.100957 |
Ejemplares similares
-
Defects in MAP1S‐mediated autophagy cause reduction in mouse lifespans especially when fibronectin is overexpressed
por: Li, Wenjiao, et al.
Publicado: (2016) -
Fast clearance of lipid droplets through MAP1S-activated autophagy suppresses clear cell renal cell carcinomas and promotes patient survival
por: Xu, Guibin, et al.
Publicado: (2015) -
Mitochondrion-associated protein LRPPRC suppresses the initiation of basal levels of autophagy via enhancing Bcl-2 stability
por: Zou, Jing, et al.
Publicado: (2013) -
Suppressor of hepatocellular carcinoma RASSF1A activates autophagy initiation and maturation
por: Li, Wenjiao, et al.
Publicado: (2018) -
Autophagy Inhibitor LRPPRC Suppresses Mitophagy through Interaction with Mitophagy Initiator Parkin
por: Zou, Jing, et al.
Publicado: (2014)