An Update on Inflamm-Aging: Mechanisms, Prevention, and Treatment

Inflamm-aging is a challenging and promising new branch of aging-related research fields that includes areas such as immunosenescence. Increasing evidence indicates that inflamm-aging is intensively associated with many aging diseases, such as Alzheimer's disease, atherosclerosis, heart disease...

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Autores principales: Xia, Shijin, Zhang, Xinyan, Zheng, Songbai, Khanabdali, Ramin, Kalionis, Bill, Wu, Junzhen, Wan, Wenbin, Tai, Xiantao
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi Publishing Corporation 2016
Materias:
Review Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4963991/
https://www.ncbi.nlm.nih.gov/pubmed/27493973
http://dx.doi.org/10.1155/2016/8426874
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author Xia, Shijin
Zhang, Xinyan
Zheng, Songbai
Khanabdali, Ramin
Kalionis, Bill
Wu, Junzhen
Wan, Wenbin
Tai, Xiantao
author_facet Xia, Shijin
Zhang, Xinyan
Zheng, Songbai
Khanabdali, Ramin
Kalionis, Bill
Wu, Junzhen
Wan, Wenbin
Tai, Xiantao
author_sort Xia, Shijin
collection PubMed
description Inflamm-aging is a challenging and promising new branch of aging-related research fields that includes areas such as immunosenescence. Increasing evidence indicates that inflamm-aging is intensively associated with many aging diseases, such as Alzheimer's disease, atherosclerosis, heart disease, type II diabetes, and cancer. Mounting studies have focused on the role of inflamm-aging in disease progression and many advances have been made in the last decade. However, the underlying mechanisms by which inflamm-aging affects pathological changes and disease development are still unclear. Here, we review studies of inflamm-aging that explore the concept, pathological features, mechanisms, intervention, and the therapeutic strategies of inflamm-aging in disease progression.
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spelling pubmed-49639912016-08-04 An Update on Inflamm-Aging: Mechanisms, Prevention, and Treatment Xia, Shijin Zhang, Xinyan Zheng, Songbai Khanabdali, Ramin Kalionis, Bill Wu, Junzhen Wan, Wenbin Tai, Xiantao J Immunol Res Review Article Inflamm-aging is a challenging and promising new branch of aging-related research fields that includes areas such as immunosenescence. Increasing evidence indicates that inflamm-aging is intensively associated with many aging diseases, such as Alzheimer's disease, atherosclerosis, heart disease, type II diabetes, and cancer. Mounting studies have focused on the role of inflamm-aging in disease progression and many advances have been made in the last decade. However, the underlying mechanisms by which inflamm-aging affects pathological changes and disease development are still unclear. Here, we review studies of inflamm-aging that explore the concept, pathological features, mechanisms, intervention, and the therapeutic strategies of inflamm-aging in disease progression. Hindawi Publishing Corporation 2016 2016-07-14 /pmc/articles/PMC4963991/ /pubmed/27493973 http://dx.doi.org/10.1155/2016/8426874 Text en Copyright © 2016 Shijin Xia et al. https://creativecommons.org/licenses/by/4.0/ This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Review Article
Xia, Shijin
Zhang, Xinyan
Zheng, Songbai
Khanabdali, Ramin
Kalionis, Bill
Wu, Junzhen
Wan, Wenbin
Tai, Xiantao
An Update on Inflamm-Aging: Mechanisms, Prevention, and Treatment
title An Update on Inflamm-Aging: Mechanisms, Prevention, and Treatment
title_full An Update on Inflamm-Aging: Mechanisms, Prevention, and Treatment
title_fullStr An Update on Inflamm-Aging: Mechanisms, Prevention, and Treatment
title_full_unstemmed An Update on Inflamm-Aging: Mechanisms, Prevention, and Treatment
title_short An Update on Inflamm-Aging: Mechanisms, Prevention, and Treatment
title_sort update on inflamm-aging: mechanisms, prevention, and treatment
topic Review Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4963991/
https://www.ncbi.nlm.nih.gov/pubmed/27493973
http://dx.doi.org/10.1155/2016/8426874
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