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Endogenous electromagnetic forces emissions during cell respiration as additional factor in cancer origin
BACKGROUND: Seven decades ago, a seminal paper by Dr. Denham Harman in (J Gerontol 11(3):298–300, 1956), introduced a theory stating that there are good reasons for assuming that endogenous irradiation in the living cells could lead to cancer via an obscure mechanism. The main purpose of this manusc...
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Formato: | Online Artículo Texto |
Lenguaje: | English |
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BioMed Central
2016
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4964086/ https://www.ncbi.nlm.nih.gov/pubmed/27471435 http://dx.doi.org/10.1186/s12935-016-0337-y |
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author | Embi, Abraham A. |
author_facet | Embi, Abraham A. |
author_sort | Embi, Abraham A. |
collection | PubMed |
description | BACKGROUND: Seven decades ago, a seminal paper by Dr. Denham Harman in (J Gerontol 11(3):298–300, 1956), introduced a theory stating that there are good reasons for assuming that endogenous irradiation in the living cells could lead to cancer via an obscure mechanism. The main purpose of this manuscript is to shed some light in said mechanism by proposing a five-step eukaryotic cell cancer triggering cycle. In other words, a new factor is introduced, namely the recently found emissions of electromagnetic forces (EMFs) as a possible causing agent in diseases, including cancer. METHODS: Introduced is an eukaryotic cell cancer inducing cycle. It includes five sequential steps of endogenous biological process that are backed by published scientific reports. RESULTS AND DISCUSSION: It is a known fact that in order to achieve homeostasis, toxic reactive oxygen species (ROS) i.e. H(2)O(2) molecules are broken down by the protein enzyme catalase. During this reaction EMFs are generated (Embi in AIS Physics 2(3):226–230, 2016). The EMFs recording breakthrough was possible due to the introduction of a novel table top microscopy technique to detect EMFs by using Prussian Blue Stain and nano-sized iron particles. There are different roots in molecular and clinical biology through which DNA damage could be programmed, EMFs emitted (during cell respiration) are herein proposed as an additional cause. |
format | Online Article Text |
id | pubmed-4964086 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2016 |
publisher | BioMed Central |
record_format | MEDLINE/PubMed |
spelling | pubmed-49640862016-07-29 Endogenous electromagnetic forces emissions during cell respiration as additional factor in cancer origin Embi, Abraham A. Cancer Cell Int Hypothesis BACKGROUND: Seven decades ago, a seminal paper by Dr. Denham Harman in (J Gerontol 11(3):298–300, 1956), introduced a theory stating that there are good reasons for assuming that endogenous irradiation in the living cells could lead to cancer via an obscure mechanism. The main purpose of this manuscript is to shed some light in said mechanism by proposing a five-step eukaryotic cell cancer triggering cycle. In other words, a new factor is introduced, namely the recently found emissions of electromagnetic forces (EMFs) as a possible causing agent in diseases, including cancer. METHODS: Introduced is an eukaryotic cell cancer inducing cycle. It includes five sequential steps of endogenous biological process that are backed by published scientific reports. RESULTS AND DISCUSSION: It is a known fact that in order to achieve homeostasis, toxic reactive oxygen species (ROS) i.e. H(2)O(2) molecules are broken down by the protein enzyme catalase. During this reaction EMFs are generated (Embi in AIS Physics 2(3):226–230, 2016). The EMFs recording breakthrough was possible due to the introduction of a novel table top microscopy technique to detect EMFs by using Prussian Blue Stain and nano-sized iron particles. There are different roots in molecular and clinical biology through which DNA damage could be programmed, EMFs emitted (during cell respiration) are herein proposed as an additional cause. BioMed Central 2016-07-28 /pmc/articles/PMC4964086/ /pubmed/27471435 http://dx.doi.org/10.1186/s12935-016-0337-y Text en © The Author(s) 2016 Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated. |
spellingShingle | Hypothesis Embi, Abraham A. Endogenous electromagnetic forces emissions during cell respiration as additional factor in cancer origin |
title | Endogenous electromagnetic forces emissions during cell respiration as additional factor in cancer origin |
title_full | Endogenous electromagnetic forces emissions during cell respiration as additional factor in cancer origin |
title_fullStr | Endogenous electromagnetic forces emissions during cell respiration as additional factor in cancer origin |
title_full_unstemmed | Endogenous electromagnetic forces emissions during cell respiration as additional factor in cancer origin |
title_short | Endogenous electromagnetic forces emissions during cell respiration as additional factor in cancer origin |
title_sort | endogenous electromagnetic forces emissions during cell respiration as additional factor in cancer origin |
topic | Hypothesis |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4964086/ https://www.ncbi.nlm.nih.gov/pubmed/27471435 http://dx.doi.org/10.1186/s12935-016-0337-y |
work_keys_str_mv | AT embiabrahama endogenouselectromagneticforcesemissionsduringcellrespirationasadditionalfactorincancerorigin |