Inhibition of microglial inflammatory responses by norepinephrine: effects on nitric oxide and interleukin-1β production

BACKGROUND: Under pathological conditions, microglia produce proinflammatory mediators which contribute to neurologic damage, and whose levels can be modulated by endogenous factors including neurotransmitters such as norepinephrine (NE). We investigated the ability of NE to suppress microglial acti...

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Detalles Bibliográficos
Autores principales: Dello Russo, Cinzia, Boullerne, Anne I, Gavrilyuk, Vitaliy, Feinstein, Douglas L
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2004
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC500870/
https://www.ncbi.nlm.nih.gov/pubmed/15285793
http://dx.doi.org/10.1186/1742-2094-1-9

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC500870/
https://www.ncbi.nlm.nih.gov/pubmed/15285793
http://dx.doi.org/10.1186/1742-2094-1-9

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