Mitochondrial dysfunction is an important cause of neurological deficits in an inflammatory model of multiple sclerosis

Neuroinflammation can cause major neurological dysfunction, without demyelination, in both multiple sclerosis (MS) and a mouse model of the disease (experimental autoimmune encephalomyelitis; EAE), but the mechanisms remain obscure. Confocal in vivo imaging of the mouse EAE spinal cord reveals that...

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Detalles Bibliográficos
Autores principales: Sadeghian, Mona, Mastrolia, Vincenzo, Rezaei Haddad, Ali, Mosley, Angelina, Mullali, Gizem, Schiza, Dimitra, Sajic, Marija, Hargreaves, Iain, Heales, Simon, Duchen, Michael R., Smith, Kenneth J.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2016
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5021937/
https://www.ncbi.nlm.nih.gov/pubmed/27624721
http://dx.doi.org/10.1038/srep33249

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