Cargando…
Involvement of Endoplasmic Reticulum Stress, Autophagy, and Apoptosis in Advanced Glycation End Products-Induced Glomerular Mesangial Cell Injury
Advanced glycation end-products (AGEs)-induced mesangial cell death is one of major causes of glomerulus dysfunction in diabetic nephropathy. Both endoplasmic reticulum (ER) stress and autophagy are adaptive responses in cells under environmental stress and participate in the renal diseases. The rol...
Autores principales: | Chiang, Chih-Kang, Wang, Ching-Chia, Lu, Tien-Fong, Huang, Kuo-How, Sheu, Meei-Ling, Liu, Shing-Hwa, Hung, Kuan-Yu |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group
2016
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5035926/ https://www.ncbi.nlm.nih.gov/pubmed/27665710 http://dx.doi.org/10.1038/srep34167 |
Ejemplares similares
-
Role of Calbindin-D28k in Diabetes-Associated Advanced Glycation End-Products-Induced Renal Proximal Tubule Cell Injury
por: Huang, Kuo-How, et al.
Publicado: (2019) -
The pathological role of advanced glycation end products-downregulated heat shock protein 60 in islet β-cell hypertrophy and dysfunction
por: Guan, Siao-Syun, et al.
Publicado: (2016) -
Activation of PI3K in response to high glucose leads to regulation of SOCS-3 and STAT1/3 signals and induction of glomerular mesangial extracellular matrix formation
por: Sheu, Meei-Ling, et al.
Publicado: (2017) -
Homocysteine induces human mesangial cell apoptosis via the involvement of autophagy and endoplasmic reticulum stress
por: Liang, Shanshan, et al.
Publicado: (2019) -
Advanced Glycation End Products Induce Peroxisome Proliferator-Activated Receptor γ Down-Regulation-Related Inflammatory Signals in Human Chondrocytes via Toll-Like Receptor-4 and Receptor for Advanced Glycation End Products
por: Chen, Ying Ju, et al.
Publicado: (2013)