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Guiding TRAIL to cancer cells through Kv10.1 potassium channel overcomes resistance to doxorubicin

Resisting cell death is one of the hallmarks of cancer, and represents a common problem resulting in ineffective cancer therapy. To overcome resistance to apoptosis, we designed an antibody-based therapy strategy using Kv10.1 as a target. Kv10.1 is a voltage-gated potassium channel, which has been i...

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Autores principales: Hartung, Franziska, Pardo, Luis A.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer Berlin Heidelberg 2016
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5045482/
https://www.ncbi.nlm.nih.gov/pubmed/27350552
http://dx.doi.org/10.1007/s00249-016-1149-7
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author Hartung, Franziska
Pardo, Luis A.
author_facet Hartung, Franziska
Pardo, Luis A.
author_sort Hartung, Franziska
collection PubMed
description Resisting cell death is one of the hallmarks of cancer, and represents a common problem resulting in ineffective cancer therapy. To overcome resistance to apoptosis, we designed an antibody-based therapy strategy using Kv10.1 as a target. Kv10.1 is a voltage-gated potassium channel, which has been identified as a tumor marker several years ago. The agent consists of a Kv10.1-specific single-chain antibody fused to the soluble tumor necrosis factor-related apoptosis-inducing ligand (scFv62-TRAIL). We combined scFv62-TRAIL with different chemotherapeutic drugs, all of which failed to induce apoptosis when used alone. In the combination, we could overcome the resistance and selectively induce apoptosis. Among the drugs, doxorubicin showed the most promising effect. Additionally, we observed improved efficacy by pre-treating the cells with doxorubicin before scFv62-TRAIL application. Expression analysis of the TRAIL death receptors suggests a doxorubicin-induced increase in the abundance of receptors as the mechanism for sensitization. Furthermore, we confirmed the anti-tumor effect and efficacy of our combination strategy in vivo in SCID mice bearing subcutaneous tumors. In conclusion, we propose a novel strategy to overcome resistance to chemotherapy in cancer cells. Doxorubicin and scFv62-TRAIL reciprocally sensitize the cells to each other, specifically in Kv10.1-positive tumor cells.
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spelling pubmed-50454822016-10-15 Guiding TRAIL to cancer cells through Kv10.1 potassium channel overcomes resistance to doxorubicin Hartung, Franziska Pardo, Luis A. Eur Biophys J Original Article Resisting cell death is one of the hallmarks of cancer, and represents a common problem resulting in ineffective cancer therapy. To overcome resistance to apoptosis, we designed an antibody-based therapy strategy using Kv10.1 as a target. Kv10.1 is a voltage-gated potassium channel, which has been identified as a tumor marker several years ago. The agent consists of a Kv10.1-specific single-chain antibody fused to the soluble tumor necrosis factor-related apoptosis-inducing ligand (scFv62-TRAIL). We combined scFv62-TRAIL with different chemotherapeutic drugs, all of which failed to induce apoptosis when used alone. In the combination, we could overcome the resistance and selectively induce apoptosis. Among the drugs, doxorubicin showed the most promising effect. Additionally, we observed improved efficacy by pre-treating the cells with doxorubicin before scFv62-TRAIL application. Expression analysis of the TRAIL death receptors suggests a doxorubicin-induced increase in the abundance of receptors as the mechanism for sensitization. Furthermore, we confirmed the anti-tumor effect and efficacy of our combination strategy in vivo in SCID mice bearing subcutaneous tumors. In conclusion, we propose a novel strategy to overcome resistance to chemotherapy in cancer cells. Doxorubicin and scFv62-TRAIL reciprocally sensitize the cells to each other, specifically in Kv10.1-positive tumor cells. Springer Berlin Heidelberg 2016-06-27 2016 /pmc/articles/PMC5045482/ /pubmed/27350552 http://dx.doi.org/10.1007/s00249-016-1149-7 Text en © The Author(s) 2016 Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.
spellingShingle Original Article
Hartung, Franziska
Pardo, Luis A.
Guiding TRAIL to cancer cells through Kv10.1 potassium channel overcomes resistance to doxorubicin
title Guiding TRAIL to cancer cells through Kv10.1 potassium channel overcomes resistance to doxorubicin
title_full Guiding TRAIL to cancer cells through Kv10.1 potassium channel overcomes resistance to doxorubicin
title_fullStr Guiding TRAIL to cancer cells through Kv10.1 potassium channel overcomes resistance to doxorubicin
title_full_unstemmed Guiding TRAIL to cancer cells through Kv10.1 potassium channel overcomes resistance to doxorubicin
title_short Guiding TRAIL to cancer cells through Kv10.1 potassium channel overcomes resistance to doxorubicin
title_sort guiding trail to cancer cells through kv10.1 potassium channel overcomes resistance to doxorubicin
topic Original Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5045482/
https://www.ncbi.nlm.nih.gov/pubmed/27350552
http://dx.doi.org/10.1007/s00249-016-1149-7
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