Cargando…
Activation of Xer-recombination at dif: structural basis of the FtsKγ–XerD interaction
Bacterial chromosomes are most often circular DNA molecules. This can produce a topological problem; a genetic crossover from homologous recombination results in dimerization of the chromosome. A chromosome dimer is lethal unless resolved. A site-specific recombination system catalyses this dimer-re...
Autores principales: | Keller, Andrew N., Xin, Yue, Boer, Stephanie, Reinhardt, Jonathan, Baker, Rachel, Arciszewska, Lidia K., Lewis, Peter J., Sherratt, David J., Löwe, Jan, Grainge, Ian |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group
2016
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5052618/ https://www.ncbi.nlm.nih.gov/pubmed/27708355 http://dx.doi.org/10.1038/srep33357 |
Ejemplares similares
-
FtsK translocation on DNA stops at XerCD-dif
por: Graham, James E., et al.
Publicado: (2010) -
Activation of XerCD-dif recombination by the FtsK DNA translocase
por: Grainge, Ian, et al.
Publicado: (2011) -
Dissection of a functional interaction between the DNA translocase, FtsK, and the XerD recombinase
por: Yates, James, et al.
Publicado: (2006) -
Asymmetric DNA requirements in Xer recombination activation by FtsK
por: Bonné, Laetitia, et al.
Publicado: (2009) -
TPM analyses reveal that FtsK contributes both to the assembly and the activation of the XerCD-dif recombination synapse
por: Diagne, Cheikh Tidiane, et al.
Publicado: (2014)